Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis (Q28511080)

GOA release 2020-03-11

5

S. Zinkel

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25 January 2017

X. M. Yin

1

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25 January 2017

K. Wang

2

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25 January 2017

A. Gross

3

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25 January 2017

Y. Zhao

4

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25 January 2017

B. Klocke

6

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25 January 2017

K. A. Roth

7

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25 January 2017

S. J. Korsmeyer

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

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25 January 2017

language of work or name

English

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publication date

26 August 1999

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25 January 2017

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25 January 2017

400

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25 January 2017

6747

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25 January 2017

886–891

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25 January 2017

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7 January 2021

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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7 January 2021

Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death

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7 January 2021

Cif (Cytochrome c efflux-inducing factor) activity is regulated by Bcl-2 and caspases and correlates with the activation of Bid.

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7 January 2021

Distinct caspase cascades are initiated in receptor-mediated and chemical-induced apoptosis.

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7 January 2021

Solution structure of BID, an intracellular amplifier of apoptotic signaling

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7 January 2021

Solution structure of the proapoptotic molecule BID: a structural basis for apoptotic agonists and antagonists

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7 January 2021

BID: a novel BH3 domain-only death agonist

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7 January 2021

BID, a proapoptotic BCL-2 family member, is localized to mouse chromosome 6 and human chromosome 22q11.

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7 January 2021

A novel domain within the 55 kd TNF receptor signals cell death

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7 January 2021

Apoptosis: telling cells their time is up.

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7 January 2021

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

1 reference

7 January 2021

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

1 reference

7 January 2021

Lethal effect of the anti-Fas antibody in mice

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7 January 2021

A combinatorial approach defines specificities of members of the caspase family and granzyme B. Functional relationships established for key mediators of apoptosis

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7 January 2021

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

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7 January 2021

A Conformational Change in Cytochromecof Apoptotic and Necrotic Cells Is Detected by Monoclonal Antibody Binding and Mimicked by Association of the Native Antigen with Synthetic Phospholipid Vesicles†

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7 January 2021

Two CD95 (APO-1/Fas) signaling pathways

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7 January 2021

DFF, a heterodimeric protein that functions downstream of caspase-3 to trigger DNA fragmentation during apoptosis

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7 January 2021

A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD

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7 January 2021

Bcl–2 protects from lethal hepatic apoptosis induced by an ant–Fas antibody in mice

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7 January 2021

A bcl-2 transgene expressed in hepatocytes protects mice from fulminant liver destruction but not from rapid death induced by anti-Fas antibody injection

1 reference

7 January 2021

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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7 January 2021

Targeted disruption of the mouse Caspase 8 gene ablates cell death induction by the TNF receptors, Fas/Apo1, and DR3 and is lethal prenatally.

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7 January 2021

Reduced apoptosis and cytochrome c-mediated caspase activation in mice lacking caspase 9

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7 January 2021

Differential requirement for caspase 9 in apoptotic pathways in vivo.

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7 January 2021

Apaf1 (CED-4 homolog) regulates programmed cell death in mammalian development

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7 January 2021

Apaf1 is required for mitochondrial pathways of apoptosis and brain development

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7 January 2021

The Bcl-2 protein family: arbiters of cell survival

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7 January 2021

In situ immunodetection of activated caspase-3 in apoptotic neurons in the developing nervous system

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7 January 2021