Redox factor-1 contributes to the regulation of progression from G0/G1 to S by PDGF in vascular smooth muscle cells (Q28566116)

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3 November 2019

title

Redox factor-1 contributes to the regulation of progression from G0/G1 to S by PDGF in vascular smooth muscle cells (English)

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3

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3 November 2019

author name string

Tongrong He

1

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3 November 2019

Papri Chatterjee

4

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3 November 2019

Neal L Weintraub

2

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3 November 2019

Dawn M Flaherty

5

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Frederick E Domann

6

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3 November 2019

Larry W Oberley

7

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publication date

1 May 2003

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American Journal of Physiology Heart and Circulatory Physiology

3 November 2019

published in

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volume

285

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issue

2

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3 November 2019

page(s)

H804-12

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Activation of the cyclin D1 gene by the E1A-associated protein p300 through AP-1 inhibits cellular apoptosis

3 November 2019

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Overexpression of human catalase inhibits proliferation and promotes apoptosis in vascular smooth muscle cells

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A c-fos/Estrogen receptor fusion protein promotes cell cycle progression and proliferation of human cancer cell lines

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Regulation of gene expression by reactive oxygen

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Redox factor-1 (Ref-1) mediates the activation of AP-1 in HeLa and NIH 3T3 cells in response to heat shock.

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AP-1 transcriptional activity is regulated by a direct association between thioredoxin and Ref-1

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Increased glutathione synthesis associated with platelet-derived growth factor stimulation of NIH3T3 fibroblasts.

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Identification of redox/repair protein Ref-1 as a potent activator of p53

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A redox mechanism controls differential DNA binding activities of hypoxia-inducible factor (HIF) 1alpha and the HIF-like factor.

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H(2)O(2)-induced O(2) production by a non-phagocytic NAD(P)H oxidase causes oxidant injury

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Involvement of tyrosine phosphorylation and protein kinase C in the activation of phospholipase D by H2O2 in Swiss 3T3 fibroblasts

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Cyclin-dependent regulation of G1 in mammalian fibroblasts.

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Smooth muscle cells and the pathogenesis of the lesions of atherosclerosis

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Requirement for generation of H2O2 for platelet-derived growth factor signal transduction

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An 'environment to nucleus' signaling system operates in B lymphocytes: redox status modulates BSAP/Pax-5 activation through Ref-1 nuclear translocation

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21 January 2018

Identifiers

DOI

2 references

31 January 2017

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3 November 2019

2 references

31 January 2017