Vascular factors altered in glucose-treated mesangial cells and diabetic glomeruli. Changes in vascular factors impair endothelial cell growth and matrix (Q28567819)

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English	Vascular factors altered in glucose-treated mesangial cells and diabetic glomeruli. Changes in vascular factors impair endothelial cell growth and matrix	scientific journal article

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scholarly article

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31 January 2017

Vascular factors altered in glucose-treated mesangial cells and diabetic glomeruli. Changes in vascular factors impair endothelial cell growth and matrix (English)

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31 January 2017

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R. Brooks Robey

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object named as

Robert B. Robey

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31 January 2017

author name string

Ashok K. Singh

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31 January 2017

Krishnamurthy P. Gudehithlu

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31 January 2017

Alfredo A. Pegoraro

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31 January 2017

Gogi K. Singh

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31 January 2017

Khaja Basheerudin

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31 January 2017

Jose A. L. Arruda

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31 January 2017

George Dunea

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language of work or name

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publication date

1 May 2004

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31 January 2017

Laboratory Investigation

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84

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597–606

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31 January 2017

Stimulation of collagen gene expression and protein synthesis in murine mesangial cells by high glucose is mediated by autocrine activation of transforming growth factor-beta

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Expression of transforming growth factor beta is elevated in human and experimental diabetic nephropathy

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Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia

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Glomerular-specific alterations of VEGF-A expression lead to distinct congenital and acquired renal diseases

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Tie-1 and tie-2 define another class of putative receptor tyrosine kinase genes expressed in early embryonic vascular system

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Distinct roles of the receptor tyrosine kinases Tie-1 and Tie-2 in blood vessel formation

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Signaling vascular morphogenesis and maintenance

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Human mesangial cells and peripheral blood mononuclear cells produce vascular permeability factor

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Mechanical stretch induces vascular permeability factor in human mesangial cells: mechanisms of signal transduction

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VEGF expression in hypoxia and hyperglycemia: reciprocal effect on branching angiogenesis in epithelial-endothelial co-cultures.

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High glucose induced VEGF expression via PKC and ERK in glomerular podocytes

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Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

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Amino acid and cDNA sequences of a vascular endothelial cell mitogen that is homologous to platelet-derived growth factor

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Isolation of angiopoietin-1, a ligand for the TIE2 receptor, by secretion-trap expression cloning

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Regulation of angiopoietin-2 mRNA levels in bovine microvascular endothelial cells by cytokines and hypoxia.

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The nucleotide sequence of the rat cytoplasmic β–actin gene

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Vascular endothelial growth factor mRNA expression in minimal change, membranous, and diabetic nephropathy demonstrated by non-isotopic in situ hybridisation

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Increased vascular endothelial growth factor levels in the vitreous of eyes with proliferative diabetic retinopathy

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Systemic hypoxia changes the organ-specific distribution of vascular endothelial growth factor and its receptors

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Inhibition of angiogenesis on glycated collagen lattices.

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VEGF(165) mediates glomerular endothelial repair

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Angiopoietin-2, a natural antagonist for Tie2 that disrupts in vivo angiogenesis

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High glucose induces DNA damage in cultured human endothelial cells

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Diabetes complications: why is glucose potentially toxic?

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Decreased degradative enzymes in mesangial cells cultured in high glucose media

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Role of angiotensin II in glucose-induced inhibition of mesangial matrix degradation

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Role of angiotensin II in diabetic nephropathy.

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High glucose decreases matrix metalloproteinase-2 activity in rat mesangial cells via transforming growth factor-beta1

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Protein tyrosine kinases expressed in glomeruli and cultured glomerular cells: Flt-1 and VEGF expression in renal mesangial cells

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Vascular permeability factor mRNA and protein expression in human kidney

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Increased renal expression of vascular endothelial growth factor (VEGF) and its receptor VEGFR-2 in experimental diabetes

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Integrin alphavbeta3-RGDS interaction mediates fibrin-induced morphological changes of glomerular endothelial cells

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Insulin stimulates the growth and tube formation of human microvascular endothelial cells through autocrine vascular endothelial growth factor

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Glucose toxicity for human endothelial cells in culture. Delayed replication, disturbed cell cycle, and accelerated death

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10.1038/LABINVEST.3700082

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31 January 2017

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31 January 2017

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Springer Nature article ID

10.1038/labinvest.3700082

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