Hyperlipidaemia impairs the circadian clock and physiological homeostasis of vascular smooth muscle cells via the suppression of Smarcd1 (Q28569433)

31 January 2017

title

Hyperlipidaemia impairs the circadian clock and physiological homeostasis of vascular smooth muscle cells via the suppression of Smarcd1 (English)

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31 January 2017

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Siyu Chen

1

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31 January 2017

Yan Ding

2

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31 January 2017

Zhao Zhang

3

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31 January 2017

Han Wang

4

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31 January 2017

Chang Liu

5

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31 January 2017

publication date

1 June 2014

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31 January 2017

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31 January 2017

volume

233

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31 January 2017

issue

2

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31 January 2017

page(s)

159–169

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Metabolism and the control of circadian rhythms

31 January 2017

cites work

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SWItch/sucrose nonfermentable (SWI/SNF) complex subunit BAF60a integrates hepatic circadian clock and energy metabolism

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Differential utilization of saturated palmitate and unsaturated oleate: evidence from cultured myotubes

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A serum shock induces circadian gene expression in mammalian tissue culture cells

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Transcriptional coactivator PGC-1alpha integrates the mammalian clock and energy metabolism

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Genome-wide coactivation analysis of PGC-1alpha identifies BAF60a as a regulator of hepatic lipid metabolism

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Identification of SWI.SNF complex subunit BAF60a as a determinant of the transactivation potential of Fos/Jun dimers

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Functional screening of FxxLF-like peptide motifs identifies SMARCD1/BAF60a as an androgen receptor cofactor that modulates TMPRSS2 expression

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Extensive and divergent circadian gene expression in liver and heart

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Circadian cycling of the mouse liver transcriptome, as revealed by cDNA microarray, is driven by the suprachiasmatic nucleus

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Coordinated transcription of key pathways in the mouse by the circadian clock

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Effect of hyperlipidemia on the expression of circadian genes in apolipoprotein E knock-out atherosclerotic mice

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A Period 2 genetic variant interacts with plasma SFA to modify plasma lipid concentrations in adults with metabolic syndrome.

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Vascular circadian rhythms in a mouse vascular smooth muscle cell line (Movas-1).

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Regulation and characteristics of vascular smooth muscle cell phenotypic diversity.

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Matrix metalloproteinase 2 and 9 dysfunction underlie vascular stiffness in circadian clock mutant mice

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21 January 2018

Identifiers

DOI

10.1002/PATH.4338

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

2043761

OpenCitations bibliographic resource ID

2043761

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

2043761

1 reference