Bax and heart mitochondria: uncoupling and inhibition of respiration without permeability transition (Q28573333)

31 January 2017

title

Bax and heart mitochondria: uncoupling and inhibition of respiration without permeability transition (English)

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31 January 2017

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Florence Appaix

1

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31 January 2017

Karen Guerrero

2

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31 January 2017

David Rampal

3

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31 January 2017

Mohamed Izikki

4

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31 January 2017

Tuuli Kaambre

5

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31 January 2017

Peeter Sikk

6

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31 January 2017

Dieter Brdiczka

7

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31 January 2017

Catherine Riva-Lavieille

8

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31 January 2017

Jose Olivares

9

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31 January 2017

Michel Longuet

10

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31 January 2017

Bruno Antonsson

11

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31 January 2017

Valdur A. Saks

12

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31 January 2017

language of work or name

English

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publication date

2 December 2002

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Biochimica et Biophysica Acta

31 January 2017

published in

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31 January 2017

volume

1556

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31 January 2017

issue

2-3

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31 January 2017

page(s)

155–167

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Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

31 January 2017

cites work

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Bax directly induces release of cytochrome c from isolated mitochondria

7 January 2021

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Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

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Cytochrome c maintains mitochondrial transmembrane potential and ATP generation after outer mitochondrial membrane permeabilization during the apoptotic process

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Studies of Energy Transport in Heart Cells. Mitochondrial Isoenzyme of Creatine Phosphokinase: Kinetic Properties and Regulatory Action of Mg2+ Ions

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Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

7 January 2021

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7 January 2021

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Expression and purification of full-length human Bax alpha.

7 January 2021

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Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria

7 January 2021

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7 January 2021

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Osmotically-induced alterations in volume and ultrastructure of mitochondria isolated from rat liver and bovine heart

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Functional consequences of the sustained or transient activation by Bax of the mitochondrial permeability transition pore

7 January 2021

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Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

7 January 2021

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Bax and Bcl-xL independently regulate apoptotic changes of yeast mitochondria that require VDAC but not adenine nucleotide translocator

7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Mitochondrial permeability transition is a central coordinating event of apoptosis

7 January 2021

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Bcl-2 and Bax regulate the channel activity of the mitochondrial adenine nucleotide translocator

7 January 2021

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Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Calphostin C-mediated translocation and integration of Bax into mitochondria induces cytochrome c release before mitochondrial dysfunction.

7 January 2021

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10.1016/S0005-2728(02)00358-4

7 January 2021

Identifiers

DOI

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