ERK and Akt signaling pathways are involved in advanced glycation end product-induced autophagy in rat vascular smooth muscle cells (Q28574192)

2

1 reference

31 January 2017

Peilin Lu

3

1 reference

31 January 2017

Jing Gao

4

1 reference

31 January 2017

Hong He

International Journal of Molecular Medicine

5

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31 January 2017

1 April 2012

1 reference

31 January 2017

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31 January 2017

29

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31 January 2017

4

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31 January 2017

613–618

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31 January 2017

describes a project that uses

ImageQuant

object named as

Image Quant

1 reference

CZ Software Mentions: A large dataset of software mentions in the biomedical literature

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Receptor for advanced glycation end products (RAGE)-mediated neurite outgrowth and activation of NF-kappaB require the cytoplasmic domain of the receptor but different downstream signaling pathways

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Combination treatment with arsenic trioxide and irradiation enhances autophagic effects in U118-MG cells through increased mitotic arrest and regulation of PI3K/Akt and ERK1/2 signaling pathways

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Oxidative stress and autophagy

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Role of advanced glycation end products (AGEs) and their receptor (RAGE) in the pathogenesis of diabetic microangiopathy

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Amino acids interfere with the ERK1/2-dependent control of macroautophagy by controlling the activation of Raf-1 in human colon cancer HT-29 cells

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The receptor for advanced glycation end products (RAGE) is a central mediator of the interaction of AGE-beta2microglobulin with human mononuclear phagocytes via an oxidant-sensitive pathway. Implications for the pathogenesis of dialysis-related amyl

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Involvement of glycation and oxidative stress in diabetic macroangiopathy

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Activation of the receptor for advanced glycation end products triggers a p21(ras)-dependent mitogen-activated protein kinase pathway regulated by oxidant stress.

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At least 2 distinct pathways generating reactive oxygen species mediate vascular cell adhesion molecule-1 induction by advanced glycation end products

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RAGE blockade stabilizes established atherosclerosis in diabetic apolipoprotein E-null mice.

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Activation of NF-kappaB and upregulation of intracellular anti-apoptotic proteins via the IGF-1/Akt signaling in human multiple myeloma cells: therapeutic implications

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Diabetes mellitus induces accelerated growth of aortic smooth muscle cells: association with overexpression of PDGF beta-receptors

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Advanced glycation endproducts-receptor interactions stimulate the growth of human pancreatic cancer cells through the induction of platelet-derived growth factor-B

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Interaction between β2-microglobulin and advanced glycation end products in the development of dialysis related-amyloidosis

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Cell composition, replication, and apoptosis in atherosclerotic plaques after 6 months of cholesterol withdrawal

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Serum levels of advanced glycation end products are increased in patients with type 2 diabetes and coronary heart disease

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Identifiers

DOI

10.3892/IJMM.2012.891

1 reference

1610073

1610073

1 reference

1 reference

1 reference