Persistent nicotine treatment potentiates amplification of the dihydrofolate reductase gene in rat lung epithelial cells as a consequence of Ras activation (Q28580057)

From Wikidata

Jump to navigation Jump to search

scientific journal article

Language	Label	Description	Also known as
English	Persistent nicotine treatment potentiates amplification of the dihydrofolate reductase gene in rat lung epithelial cells as a consequence of Ras activation	scientific journal article

Statements

scholarly article

1 reference

31 January 2017

Persistent nicotine treatment potentiates amplification of the dihydrofolate reductase gene in rat lung epithelial cells as a consequence of Ras activation (English)

1 reference

31 January 2017

0 references

author name string

Jinjin Guo

1

1 reference

31 January 2017

Michelle Chu

2

1 reference

31 January 2017

Tetteh Abbeyquaye

3

1 reference

31 January 2017

Chang-Yan Chen

4

1 reference

31 January 2017

language of work or name

0 references

publication date

26 August 2005

1 reference

31 January 2017

Journal of Biological Chemistry

1 reference

31 January 2017

280

1 reference

31 January 2017

34

1 reference

31 January 2017

30422–30431

1 reference

31 January 2017

Drug therapy. Pharmacologic aspects of cigarette smoking and nicotine addiction

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Opioid and nicotine receptors affect growth regulation of human lung cancer cell lines

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Effects of nicotine on the nucleus accumbens and similarity to those of addictive drugs

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Nicotine stimulates angiogenesis and promotes tumor growth and atherosclerosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Signalling pathways involved in nicotine regulation of apoptosis of human lung cancer cells.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Mechanisms of mitogen-activated protein kinase activation by nicotine in small-cell lung carcinoma cells

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Nicotine exposure and bronchial epithelial cell nicotinic acetylcholine receptor expression in the pathogenesis of lung cancer

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Long-term exposure to nicotine, via ras pathway, induces cyclin D1 to stimulate G1 cell cycle transition

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

A functional role for nicotine in Bcl2 phosphorylation and suppression of apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Rapid Akt activation by nicotine and a tobacco carcinogen modulates the phenotype of normal human airway epithelial cells

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Cancer cell cycles

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Wild-type p53 restores cell cycle control and inhibits gene amplification in cells with mutant p53 alleles.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Enhancement of methotrexate resistance and dihydrofolate reductase gene amplification by treatment of mouse 3T6 cells with hydroxyurea

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Correlation of genetic instability and apoptosis in the presence of oncogenic Ki-Ras

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

The human Ha-ras oncogene induces genomic instability in murine fibroblasts within one cell cycle

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Gene amplification in a p53-deficient cell line requires cell cycle progression under conditions that generate DNA breakage

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Ras effectors and their role in mitogenesis and oncogenesis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Cell cycle: routine role for Ras.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Oncogenic ras mediates apoptosis in response to protein kinase C inhibition through the generation of reactive oxygen species

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Down-regulation of platelet-derived growth factor receptor expression during terminal differentiation of 3T3-L1 pre-adipocyte fibroblasts

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Expression of Rap 1 suppresses genomic instability of H-ras transformed mouse fibroblasts

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Somatic activation of the K-ras oncogene causes early onset lung cancer in mice

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Defects in a cell cycle checkpoint may be responsible for the genomic instability of cancer cells

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Cell cycle control and cancer

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Doing the right thing: feedback control and p53.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

p53 function and dysfunction

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

P53 is necessary for the apoptotic response mediated by a transient increase of Ras activity

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Requirement of NF-kappaB activation to suppress p53-independent apoptosis induced by oncogenic Ras.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

The retinoblastoma protein: more than a tumor suppressor

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Acute mutation of retinoblastoma gene function is sufficient for cell cycle re-entry

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Regulation and mechanisms of gene amplification

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Differential effects of phorbol ester on growth and protein kinase C isoenzyme regulation in human hepatoma Hep3B cells

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Studies and perspectives of protein kinase C.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Repair of oxidative damage to DNA: enzymology and biology

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Synergy between the Mos/mitogen-activated protein kinase pathway and loss of p53 function in transformation and chromosome instability

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

A Ras activation pathway dependent on Syk phosphorylation of protein kinase C

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Cancer statistics, 1998.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M504688200

21 January 2018

Identifiers

10.1074/JBC.M504688200

1 reference

31 January 2017

1 reference

31 January 2017

Sitelinks

Wikipedia(0 entries)

Wikibooks(0 entries)

Wikinews(0 entries)

Wikiquote(0 entries)

Wikisource(0 entries)

Wikiversity(0 entries)

Wikivoyage(0 entries)

Wiktionary(0 entries)

Multilingual sites(0 entries)

Retrieved from "https://www.wikidata.org/w/index.php?title=Q28580057&oldid=1410176923"