Transgenic rescue of defective Cd36 ameliorates insulin resistance in spontaneously hypertensive rats (Q28582143)

2

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31 January 2017

V. Zidek

3

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31 January 2017

A. Musilova

4

1 reference

31 January 2017

V. Kren

5

1 reference

31 January 2017

L. Kazdova

6

1 reference

31 January 2017

T. J. Aitman

7

1 reference

31 January 2017

A. M. Glazier

8

1 reference

31 January 2017

A. Ibrahimi

9

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31 January 2017

N. A. Abumrad

10

1 reference

31 January 2017

N. Qi

11

1 reference

31 January 2017

J. M. Wang

12

1 reference

31 January 2017

E. M. St Lezin

13

1 reference

31 January 2017

T. W. Kurtz

Quantitative trait loci for cellular defects in glucose and fatty acid metabolism in hypertensive rats

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31 January 2017

language of work or name

English

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publication date

1 February 2001

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31 January 2017

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31 January 2017

27

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31 January 2017

2

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31 January 2017

156–158

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31 January 2017

1 reference

7 January 2021

Identification of Cd36 (Fat) as an insulin-resistance gene causing defective fatty acid and glucose metabolism in hypertensive rats.

1 reference

7 January 2021

Genetics of Cd36 and the clustering of multiple cardiovascular risk factors in spontaneous hypertension

1 reference

7 January 2021

A null mutation in murine CD36 reveals an important role in fatty acid and lipoprotein metabolism.

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7 January 2021

Absence of Cd36 mutation in the original spontaneously hypertensive rats with insulin resistance.

1 reference

7 January 2021

Role of fatty acids in the pathogenesis of insulin resistance and NIDDM.

1 reference

7 January 2021

Cellular mechanisms of insulin resistance

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7 January 2021

Expression of the CD36 homolog (FAT) in fibroblast cells: effects on fatty acid transport

1 reference

7 January 2021