Total and mitochondrial nitrosative stress, decreased brain-derived neurotrophic factor (BDNF) levels and glutamate uptake, and evidence of endoplasmic reticulum stress in the hippocampus of vitamin A-treated rats (Q28582962)

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English	Total and mitochondrial nitrosative stress, decreased brain-derived neurotrophic factor (BDNF) levels and glutamate uptake, and evidence of endoplasmic reticulum stress in the hippocampus of vitamin A-treated rats	scientific journal article

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scholarly article

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31 January 2017

Total and mitochondrial nitrosative stress, decreased brain-derived neurotrophic factor (BDNF) levels and glutamate uptake, and evidence of endoplasmic reticulum stress in the hippocampus of vitamin A-treated rats (English)

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31 January 2017

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endoplasmic reticulum

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based on heuristic

inferred from title

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based on heuristic

inferred from title

Diogo L de Oliveira

object named as

Diogo Losch de Oliveira

5

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object named as

Laura Stertz

3

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Marcos Roberto de Oliveira

object named as

Marcos Roberto de Oliveira

1

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Gabriel R. Fries

object named as

Gabriel Rodrigo Fries

4

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Flávio Kapczinski

6

object named as

Flávio Kapczinski

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31 January 2017

author name string

Ricardo Fagundes da Rocha

2

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31 January 2017

José Cláudio Fonseca Moreira

7

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31 January 2017

publication date

1 March 2011

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31 January 2017

Neurochemical Research

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31 January 2017

36

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31 January 2017

3

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31 January 2017

506–517

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31 January 2017

https://scigraph.springernature.com/pub.10.1007/s11064-010-0372-3

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Role of retinoid signalling in the adult brain

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Vitamin A and preterm infants: what we know, what we don't know, and what we need to know

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Vitamin neurotoxicity

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Oxidative stress in the hippocampus, anxiety-like behavior and decreased locomotory and exploratory activity of adult rats: effects of sub acute vitamin A supplementation at therapeutic doses.

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Vitamin A supplementation induces a prooxidative state in the striatum and impairs locomotory and exploratory activity of adult rats.

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Acute and chronic vitamin A supplementation at therapeutic doses induces oxidative stress in submitochondrial particles isolated from cerebral cortex and cerebellum of adult rats.

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Therapeutic vitamin A doses increase the levels of markers of oxidative insult in substantia nigra and decrease locomotory and exploratory activity in rats after acute and chronic supplementation.

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Vitamin A supplementation at pharmacological doses induces nitrosative stress on the hypothalamus of adult Wistar rats.

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Vitamin A supplementation at clinical doses induces a dysfunction in the redox and bioenergetics states, but did change neither caspases activities nor TNF-alpha levels in the frontal cortex of adult Wistar rats

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Tissue sulfhydryl groups

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Neurotrophins as synaptic modulators

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BDNF increases rat brain mitochondrial respiratory coupling at complex I, but not complex II

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Protein tyrosine nitration--functional alteration or just a biomarker?

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Peroxynitrite reactivity with amino acids and proteins

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Nitric oxide in the central nervous system: neuroprotection versus neurotoxicity

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Oxidative stress and neurodegeneration: where are we now?

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Microtubule dysfunction by posttranslational nitrotyrosination of alpha-tubulin: a nitric oxide-dependent mechanism of cellular injury

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Alpha-synuclein and neurodegenerative diseases

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Proteasomal inhibition by alpha-synuclein filaments and oligomers.

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alpha-synuclein promotes mitochondrial deficit and oxidative stress

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Mutant and wild-type alpha-synuclein interact with mitochondrial cytochrome C oxidase

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Dopamine in neurotoxicity and neuroprotection: what do D2 receptors have to do with it?

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Pathogenesis of Parkinson's disease: dopamine, vesicles and alpha-synuclein

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Calpain inhibition but not reticulum endoplasmic stress preconditioning protects rat kidneys from p-aminophenol toxicity.

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10.1007/S11064-010-0372-3

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31 January 2017

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31 January 2017

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