The bile acid receptor GPBAR-1 (TGR5) modulates integrity of intestinal barrier and immune response to experimental colitis (Q28590901)

GOA release 2020-03-11

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Sabrina Cipriani

1

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31 January 2017

Andrea Mencarelli

2

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31 January 2017

Maria Giovanna Chini

3

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31 January 2017

Eleonora Distrutti

4

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31 January 2017

Barbara Renga

5

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31 January 2017

Giuseppe Bifulco

6

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31 January 2017

Franco Baldelli

7

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31 January 2017

Annibale Donini

8

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31 January 2017

Stefano Fiorucci

Creative Commons Attribution 4.0 International

9

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31 January 2017

language of work or name

English

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publication date

1 January 2011

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31 January 2017

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31 January 2017

6

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31 January 2017

10

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31 January 2017

e25637

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31 January 2017

start time

27 October 2011

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April 2022 Public Data File from Crossref

copyright status

copyrighted

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Mutational characterization of the bile acid receptor TGR5 in primary sclerosing cholangitis

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Expression and function of the bile acid receptor GpBAR1 (TGR5) in the murine enteric nervous system.

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Expression and function of the bile acid receptor TGR5 in Kupffer cells

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Bile acids modulate tight junction structure and barrier function of Caco-2 monolayers via EGFR activation.

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Importance of innate immunity and collagen binding integrin alpha1beta1 in TNBS-induced colitis

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Novel potent and selective bile acid derivatives as TGR5 agonists: biological screening, structure-activity relationships, and molecular modeling studies

21 January 2018

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Nongenomic actions of bile acids. Synthesis and preliminary characterization of 23- and 6,23-alkyl-substituted bile acid derivatives as selective modulators for the G-protein coupled receptor TGR5.

21 January 2018

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Effect of colonic bacterial metabolites on Caco-2 cell paracellular permeability in vitro.

21 January 2018

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Intestinal permeability is reduced and IL-10 levels are increased in septic IL-6 knockout mice

21 January 2018

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Anti-hyperglycemic activity of a TGR5 agonist isolated from Olea europaea

27 November 2018

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PubMed

https://pubmed.ncbi.nlm.nih.gov/22046243

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1371/JOURNAL.PONE.0025637

1 reference

4618724

ADS bibcode

2011PLoSO...625637C

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4618724

1 reference

1 reference

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