Lsh, a SNF2 family member, is required for normal murine development (Q28591969)

31 January 2017

title

Lsh, a SNF2 family member, is required for normal murine development (English)

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Helicase, lymphoid specific

31 January 2017

main subject

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GOA release 2020-03-11

author name string

T. M. Geiman

1

1 reference

31 January 2017

L. Tessarollo

2

1 reference

31 January 2017

M. R. Anver

3

1 reference

31 January 2017

J. B. Kopp

4

1 reference

31 January 2017

J. M. Ward

5

1 reference

31 January 2017

K. Muegge

6

1 reference

31 January 2017

language of work or name

English

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publication date

3 May 2001

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Biochimica et Biophysica Acta

31 January 2017

published in

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31 January 2017

volume

1526

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31 January 2017

issue

2

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31 January 2017

page(s)

211–220

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A novel putative helicase produced in early murine lymphocytes.

31 January 2017

cites work

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Helicases: amino acid sequence comparisons and structure-function relationships

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DNA helicases in inherited human disorders

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The SNF/SWI family of global transcriptional activators

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Evolution of the SNF2 family of proteins: subfamilies with distinct sequences and functions

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Nucleosome movement by CHRAC and ISWI without disruption or trans-displacement of the histone octamer.

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ATP-dependent histone octamer sliding mediated by the chromatin remodeling complex NURF.

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Cloning of human and bovine homologs of SNF2/SWI2: a global activator of transcription in yeast S. cerevisiae

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A human homologue of Saccharomyces cerevisiae SNF2/SWI2 and Drosophila brm genes potentiates transcriptional activation by the glucocorticoid receptor

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Characterization of the CHD family of proteins

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Characterization of gene expression, genomic structure, and chromosomal localization of Hells (Lsh).

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Diversity and specialization of mammalian SWI/SNF complexes

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Lsh, an SNF2/helicase family member, is required for proliferation of mature T lymphocytes

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7 January 2021

BRG1 contains a conserved domain of the SWI2/SNF2 family necessary for normal mitotic growth and transcription

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7 January 2021

The yeast SNF2/SWI2 protein has DNA-stimulated ATPase activity required for transcriptional activation

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Promoter targeting and chromatin remodeling by the SWI/SNF complex

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7 January 2021

Characterization of an Arabidopsis thaliana DNA hypomethylation mutant

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7 January 2021

brahma: a regulator of Drosophila homeotic genes structurally related to the yeast transcriptional activator SNF2/SWI2

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7 January 2021

dMi-2, a hunchback-interacting protein that functions in polycomb repression

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The ISWI chromatin-remodeling protein is required for gene expression and the maintenance of higher order chromatin structure in vivo

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Chromosome tangling and breakage at anaphase result from mutations in lodestar, a Drosophila gene encoding a putative nucleoside triphosphate-binding protein

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Mot1, a global repressor of RNA polymerase II transcription, inhibits TBP binding to DNA by an ATP-dependent mechanism.

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Altered control of cellular proliferation in the absence of mammalian brahma (SNF2alpha).

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A Brg1 null mutation in the mouse reveals functional differences among mammalian SWI/SNF complexes

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7 January 2021

Disruption of mouse RAD54 reduces ionizing radiation resistance and homologous recombination.

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7 January 2021

Skeletal dysplasias, growth retardation, reduced postnatal survival, and impaired fertility in mice lacking the SNF2/SWI2 family member ETL1.

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7 January 2021

A C/EBP beta isoform recruits the SWI/SNF complex to activate myeloid genes

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7 January 2021

Targeting a SWI/SNF-related chromatin remodeling complex to the beta-globin promoter in erythroid cells

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7 January 2021

Enhanced apoptotic cell death of renal epithelial cells in mice lacking transcription factor AP-2beta

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7 January 2021

Mi-2 complex couples DNA methylation to chromatin remodelling and histone deacetylation

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The DNA methylation locus DDM1 is required for maintenance of gene silencing in Arabidopsis

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7 January 2021

Maintenance of genomic methylation requires a SWI2/SNF2-like protein

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7 January 2021

10.1016/S0304-4165(01)00129-5

Identifiers

DOI

0 references

1 reference