Bisphenol a exposure causes meiotic aneuploidy in the female mouse (Q29615674)

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=12676084

30 April 2017

author name string

Hunt PA

1

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30 April 2017

Koehler KE

2

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Susiarjo M

3

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Hodges CA

4

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30 April 2017

Ilagan A

5

1 reference

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Voigt RC

6

1 reference

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Thomas S

7

1 reference

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Thomas BF

8

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Hassold TJ

9

1 reference

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=12676084

30 April 2017

language of work or name

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30 April 2017

publication date

1 April 2003

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30 April 2017

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volume

13

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page(s)

546-53

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issue

7

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To err (meiotically) is human: the genesis of human aneuploidy

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Experimental evidence that changes in oocyte growth influence meiotic chromosome segregation.

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The meiotic competence of in-vitro matured human oocytes is influenced by donor age: evidence that folliculogenesis is compromised in the reproductively aged ovary.

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The xenoestrogen bisphenol A induces growth, differentiation, and c-fos gene expression in the female reproductive tract

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Estrogenic activity of octylphenol, nonylphenol, bisphenol A and methoxychlor in rats.

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Relative binding affinity-serum modified access (RBA-SMA) assay predicts the relative in vivo bioactivity of the xenoestrogens bisphenol A and octylphenol

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Determination of bisphenol A and related aromatic compounds released from bis-GMA-based composites and sealants by high performance liquid chromatography

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Toxicokinetics of bisphenol A in female DA/Han rats after a single i.v. and oral administration

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Pharmacokinetic disposition and tissue distribution of bisphenol A in rats after intravenous administration.

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A physiologically based approach to the study of bisphenol A and other estrogenic chemicals on the size of reproductive organs, daily sperm production, and behavior

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Reproductive malformation of the male offspring following maternal exposure to estrogenic chemicals

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In utero exposure to low doses of bisphenol A lead to long-term deleterious effects in the vagina.

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Developmental effects of perinatal exposure to bisphenol-A and diethylstilbestrol on reproductive organs in female mice.

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Exposure to bisphenol A advances puberty

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Perinatal exposure to low doses of bisphenol A affects body weight, patterns of estrous cyclicity, and plasma LH levels

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Interference with microtubules and induction of micronuclei in vitro by various bisphenols.

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Bisphenol-A induces cellular transformation, aneuploidy and DNA adduct formation in cultured Syrian hamster embryo cells

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7 January 2021

Induction of multiple microtubule-organizing centers, multipolar spindles and multipolar division in cultured V79 cells exposed to diethylstilbestrol, estradiol-17beta and bisphenol A

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