Interleukins 1beta and 6 but not transforming growth factor-beta are essential for the differentiation of interleukin 17-producing human T helper cells (Q29618163)

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scientific article (publication date: September 2007)

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English	Interleukins 1beta and 6 but not transforming growth factor-beta are essential for the differentiation of interleukin 17-producing human T helper cells	scientific article (publication date: September 2007)

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3 January 2020

Interleukins 1beta and 6 but not transforming growth factor-beta are essential for the differentiation of interleukin 17-producing human T helper cells (English)

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Eva Acosta Rodríguez

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Federica Sallusto

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Federica Sallusto

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3 January 2020

Antonio Lanzavecchia

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Antonio Lanzavecchia

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author name string

Giorgio Napolitani

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5 August 2007

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3 January 2020

Nature Immunology

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942-949

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3 January 2020

A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17

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Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages

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IL-17 family cytokines and the expanding diversity of effector T cell lineages

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Interleukin-23 promotes a distinct CD4 T cell activation state characterized by the production of interleukin-17

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TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

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Transforming growth factor-beta induces development of the T(H)17 lineage

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Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

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Interleukin-2 signaling via STAT5 constrains T helper 17 cell generation

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The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

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STAT3 regulates cytokine-mediated generation of inflammatory helper T cells

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Surface phenotype and antigenic specificity of human interleukin 17-producing T helper memory cells

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Langerhans cells arise from monocytes in vivo

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