Endothelial-to-mesenchymal transition contributes to cardiac fibrosis (Q29620566)

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Zeisberg EM

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Tarnavski O

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Zeisberg M

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Dorfman AL

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McMullen JR

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Gustafsson E

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Chandraker A

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Yuan X

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Pu WT

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Roberts AB

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Neilson EG

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Sayegh MH

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Izumo S

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Kalluri R

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language of work or name

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publication date

August 2007

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volume

13

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page(s)

952-61

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issue

8

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Fibrillar collagen and myocardial stiffness in the intact hypertrophied rat left ventricle

30 April 2017

cites work

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Phosphoinositide 3-kinase(p110α) plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy

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Early induction of transforming growth factor-beta via angiotensin II type 1 receptors contributes to cardiac fibrosis induced by long-term blockade of nitric oxide synthesis in rats

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Bone morphogenic protein-7 inhibits progression of chronic renal fibrosis associated with two genetic mouse models

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Impaired recruitment of bone-marrow-derived endothelial and hematopoietic precursor cells blocks tumor angiogenesis and growth

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Lack of NF-kappaB1 (p105/p50) attenuates unloading-induced downregulation of PPARalpha and PPARalpha-regulated gene expression in rodent heart

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BMP-7 counteracts TGF-beta1-induced epithelial-to-mesenchymal transition and reverses chronic renal injury

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Morphogenesis of the right ventricle requires myocardial expression of Gata4

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Identifiers

2 references

30 April 2017

Consolidated OpenCitations Corpus – April 2017

Dimensions Publication ID

508213

1050416284

0 references

508213

1 reference

Consolidated OpenCitations Corpus – April 2017

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=17660828

2 references

30 April 2017

Consolidated OpenCitations Corpus – April 2017