The p7 protein of hepatitis C virus forms an ion channel that is blocked by the antiviral drug, Amantadine (Q29620670)

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Rowlands DJ

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Stephen D.C. Griffin

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Griffin SD

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30 April 2017

Beales LP

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Clarke DS

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Worsfold O

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Evans SD

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Jaeger J

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Harris MP

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Lucy P. Beales

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30 April 2017

publication date

30 January 2003

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30 April 2017

21 December 2002

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volume

535

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page(s)

34-8

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34-38

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1-3

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=12560074

E2-p7 region of the bovine viral diarrhea virus polyprotein: processing and functional studies

30 April 2017

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The transmembrane domain of influenza A M2 protein forms amantadine-sensitive proton channels in planar lipid bilayers

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Identification of the major, parenteral non-A, non-B hepatitis agent (hepatitis C virus) using a recombinant cDNA approach

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Predictors of sustained response, relapse and no response in patients with chronic hepatitis C treated with interferon-alpha

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Subcellular Localization and Topology of the p7 Polypeptide of Hepatitis C Virus

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Coxsackievirus protein 2B modifies endoplasmic reticulum membrane and plasma membrane permeability and facilitates virus release.

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Influenza virus M2 protein has ion channel activity

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The NB protein is an integral component of the membrane of influenza B virus

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Transcripts of a chimeric cDNA clone of hepatitis C virus genotype 1b are infectious in vivo.

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Single-step purification of polypeptides expressed in Escherichia coli as fusions with glutathione S-transferase

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Activation of the M2 ion channel of influenza virus: a role for the transmembrane domain histidine residue

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Protonation of histidine and histidine-tryptophan interaction in the activation of the M2 ion channel from influenza a virus

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In vitro effect of amantadine and interferon alpha-2a on hepatitis C virus markers in cultured peripheral blood mononuclear cells from hepatitis C virus-infected patients

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Clearance kinetics of hepatitis C virus under different antiviral therapies

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Triple antiviral therapy as a new option for patients with interferon nonresponsive chronic hepatitis C.

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Amantadine and rimantadine have no direct inhibitory effects against hepatitis C viral protease, helicase, ATPase, polymerase, and internal ribosomal entry site-mediated translation

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The molecular basis of the specific anti-influenza action of amantadine

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Molecular virology of hepatitis C virus

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Membrane permeabilization by poliovirus proteins 2B and 2BC

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Ion channel activity of influenza A virus M2 protein: characterization of the amantadine block

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Treatment of chronic hepatitis C with amantadine

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Processing in the hepatitis C virus E2-NS2 region: identification of p7 and two distinct E2-specific products with different C termini

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