MicroRNA-223 regulates Glut4 expression and cardiomyocyte glucose metabolism (Q29871002)

9 May 2017

MicroRNA-223 regulates Glut4 expression and cardiomyocyte glucose metabolism (English)

1 reference

9 May 2017

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Han Lu

1

1 reference

PubMed ID

20080987

9 May 2017

Rachel J Buchan

series ordinal

2

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PubMed ID

20080987

9 May 2017

Stuart A Cook

series ordinal

3

1 reference

9 May 2017

15 January 2010

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9 May 2017

Cardiovascular Research

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9 May 2017

86

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9 May 2017

3

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9 May 2017

410-420

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Cardiac and skeletal muscle insulin resistance in patients with coronary heart disease. A study with positron emission tomography

9 May 2017

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21 January 2018

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Emerging role of microRNAs in cardiovascular biology.

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MicroRNAs control gene expression: importance for cardiac development and pathophysiology.

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21 January 2018

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Myogenic factors that regulate expression of muscle-specific microRNAs

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MicroRNA-133 controls cardiac hypertrophy

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MicroRNAs modulate hematopoietic lineage differentiation

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Type 2 diabetes, mitochondrial biology and the heart

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Oxidative stress induces GLUT4 translocation by activation of PI3-K/Akt and dual AMPK kinase in cardiac myocytes

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A signature pattern of stress-responsive microRNAs that can evoke cardiac hypertrophy and heart failure

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Control of stress-dependent cardiac growth and gene expression by a microRNA

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MicroRNA-21 contributes to myocardial disease by stimulating MAP kinase signalling in fibroblasts

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Dysregulation of microRNAs after myocardial infarction reveals a role of miR-29 in cardiac fibrosis

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Effects of glucose-insulin-potassium infusion on ST-elevation myocardial infarction in patients treated with thrombolytic therapy

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No-flow ischemia inhibits insulin signaling in heart by decreasing intracellular pH.

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Insulin and ischemia stimulate glycolysis by acting on the same targets through different and opposing signaling pathways

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The impact of microRNAs on protein output

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Switching from repression to activation: microRNAs can up-regulate translation

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