Phagosomal signaling by Borrelia burgdorferi in human monocytes involves Toll-like receptor (TLR) 2 and TLR8 cooperativity and TLR8-mediated induction of IFN-beta (Q30498311)

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English	Phagosomal signaling by Borrelia burgdorferi in human monocytes involves Toll-like receptor (TLR) 2 and TLR8 cooperativity and TLR8-mediated induction of IFN-beta	scientific article

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scholarly article

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22 January 2020

Phagosomal signaling by Borrelia burgdorferi in human monocytes involves Toll-like receptor (TLR) 2 and TLR8 cooperativity and TLR8-mediated induction of IFN-beta (English)

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22 January 2020

Borrelia burgdorferi

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toll-like receptor

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5

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22 January 2020

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Jorge L Cervantes

1

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22 January 2020

Star M Dunham-Ems

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22 January 2020

Carson J La Vake

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22 January 2020

Mary M Petzke

4

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Timothy J Sellati

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22 January 2020

Justin D Radolf

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22 January 2020

Juan C Salazar

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14 February 2011

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22 January 2020

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Proceedings of the National Academy of Sciences of the United States of America

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108

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9

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22 January 2020

3683-3688

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Genetic modulation of TLR8 response following bacterial phagocytosis.

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The Lyme disease spirochete Borrelia burgdorferi utilizes multiple ligands, including RNA, for interferon regulatory factor 3-dependent induction of type I interferon-responsive genes

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A critical role for type I IFN in arthritis development following Borrelia burgdorferi infection of mice

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Group A streptococcus activates type I interferon production and MyD88-dependent signaling without involvement of TLR2, TLR4, and TLR9.

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Phagocytosis of Borrelia burgdorferi, the Lyme disease spirochete, potentiates innate immune activation and induces apoptosis in human monocytes

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Phagocytosis of Borrelia burgdorferi and Treponema pallidum potentiates innate immune activation and induces gamma interferon production

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Murine dendritic cell type I IFN production induced by human IgG-RNA immune complexes is IFN regulatory factor (IRF)5 and IRF7 dependent and is required for IL-6 production

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10.1073/PNAS.1013776108

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2011PNAS..108.3683C

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22 January 2020

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