Substance P antagonist blocks leakage and reduces activation of cytokine-stimulated rat brain endothelium. (Q31119677)

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15 December 2019

title

Substance P antagonist blocks leakage and reduces activation of cytokine-stimulated rat brain endothelium (English)

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15 December 2019

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4

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15 December 2019

author name string

Pasquale Annunziata

series ordinal

1

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15 December 2019

Chiara Cioni

series ordinal

2

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15 December 2019

Riccardo Santonini

series ordinal

3

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15 December 2019

publication date

1 October 2002

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Journal of Neuroimmunology

15 December 2019

published in

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15 December 2019

volume

131

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15 December 2019

page(s)

41-49

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15 December 2019

issue

1-2

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Inflammatory mediators and modulation of blood-brain barrier permeability

15 December 2019

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HIV-1 gp120 increases the permeability of rat brain endothelium cultures by a mechanism involving substance P.

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Recombinant human tumor necrosis factor alpha constricts pial arterioles and increases blood-brain barrier permeability in newborn piglets

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The pharmacological profile of a substance P (SP) antagonist. Evidence for the existence of subpopulations of SP receptors

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Endothelium-dependent contraction induced by substance P in canine cerebral arteries: involvement of NK1 receptors and thromboxane A2

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7 January 2021

Substance P promotes lymphocyte-endothelial cell adhesion preferentially via LFA-1/ICAM-1 interactions

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7 January 2021

ICAM-1-dependent pathway is not critically involved in the inflammatory process of autoimmune encephalomyelitis or in cytokine-induced inflammation of the central nervous system

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7 January 2021

Upregulation of intercellular adhesion molecule-1 (ICAM-1) expression in primary cultures of human brain microvessel endothelial cells by cytokines and lipopolysaccharide

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7 January 2021

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Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12458035%20AND%20SRC:MED&resulttype=core&format=json

15 December 2019

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