Dual role of glutamatergic neurotransmission on amyloid beta(1-42) aggregation and neurotoxicity in embryonic avian retina (Q32141817)

6 July 2017

Dual role of glutamatergic neurotransmission on amyloid beta(1-42) aggregation and neurotoxicity in embryonic avian retina (English)

1 reference

6 July 2017

1 reference

1 reference

P R Louzada

1

1 reference

6 July 2017

A C Paula Lima

series ordinal

2

1 reference

6 July 2017

F G de Mello

series ordinal

3

1 reference

6 July 2017

S T Ferreira

series ordinal

4

1 reference

6 July 2017

language of work or name

English

0 references

publication date

1 March 2001

1 reference

6 July 2017

1 reference

6 July 2017

301

1 reference

6 July 2017

1

1 reference

6 July 2017

59-63

1 reference

Beta-amyloid-induced apoptosis of cerebellar granule cells and cortical neurons: exacerbation by selective inhibition of group I metabotropic glutamate receptors.

6 July 2017

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

Stable complexes involving acetylcholinesterase and amyloid-beta peptide change the biochemical properties of the enzyme and increase the neurotoxicity of Alzheimer's fibrils.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

Dramatic aggregation of Alzheimer abeta by Cu(II) is induced by conditions representing physiological acidosis.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

Selective blockade of metabotropic glutamate receptor subtype 5 is neuroprotective

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

beta-Amyloid neurotoxicity in human cortical culture is not mediated by excitotoxins

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

Cerebral beta amyloid angiopathy is a risk factor for cerebral ischemic infarction. A case control study in human brain biopsies.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

Neurodegeneration mediated by glutamate and beta-amyloid peptide: a comparison and possible interaction

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

beta-amyloid neurotoxicity is mediated by a glutamate-triggered excitotoxic cascade in rat nucleus basalis

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

N-Methyl-D-aspartate receptor antagonist MK-801 and radical scavengers protect cholinergic nucleus basalis neurons against beta-amyloid neurotoxicity.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

A new compound (AZ36041) promotes the survival of the neurons and reduces neurotoxicity of Alzheimer's beta-amyloid protein

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

A model for beta-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: relevance to Alzheimer disease

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

Alzheimer's disease amyloid beta-protein forms Zn(2+)-sensitive, cation-selective channels across excised membrane patches from hypothalamic neurons

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

Carboxyl-terminal fragment of Alzheimer's APP destabilizes calcium homeostasis and renders neuronal cells vulnerable to excitotoxicity.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

Beta-amyloid protein increases the vulnerability of cultured cortical neurons to excitotoxic damage

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

In vivo neurotoxicity of beta-amyloid [beta(1-40)] and the beta(25-35) fragment

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

Amyloid beta-peptide inhibits Na+-dependent glutamate uptake

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

Beta-amyloid neurotoxicity requires fibril formation and is inhibited by congo red.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

Calcium-destabilizing and neurodegenerative effects of aggregated beta-amyloid peptide are attenuated by basic FGF.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

Glutamate release from microglia via glutamate transporter is enhanced by amyloid-beta peptide

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

Neurodegeneration induced by beta-amyloid peptides in vitro: the role of peptide assembly state

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

In vitro aging of beta-amyloid protein causes peptide aggregation and neurotoxicity

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

1 reference

7 January 2021

Transient NMDA receptor inactivation provides long-term protection to cultured cortical neurons from a variety of death signals

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2801%2901585-3

7 January 2021

10.1016/S0304-3940(01)01585-3

Identifiers

DOI

1 reference

6 July 2017

1 reference