Proteomic analysis of brain proteins in the gracile axonal dystrophy (gad) mouse, a syndrome that emanates from dysfunctional ubiquitin carboxyl-terminal hydrolase L-1, reveals oxidation of key proteins. (Q33199152)
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scientific article
| Language | Label | Description | Also known as |
|---|---|---|---|
| English | Proteomic analysis of brain proteins in the gracile axonal dystrophy (gad) mouse, a syndrome that emanates from dysfunctional ubiquitin carboxyl-terminal hydrolase L-1, reveals oxidation of key proteins. |
scientific article |
Statements
Proteomic analysis of brain proteins in the gracile axonal dystrophy (gad) mouse, a syndrome that emanates from dysfunctional ubiquitin carboxyl-terminal hydrolase L-1, reveals oxidation of key proteins (English)
D Allan Butterfield
1 reference
Alessandra Castegna
Jon Klein
Bert C Lynn
Yu-Lai Wang
Hitoshi Osaka
Keiji Wada
Visith Thongboonkerd
D Allan Butterfield
1 March 2004