Fibrillar beta-amyloid peptide Abeta1-40 activates microglial proliferation via stimulating TNF-alpha release and H2O2 derived from NADPH oxidase: a cell culture study (Q33256753)

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English	Fibrillar beta-amyloid peptide Abeta1-40 activates microglial proliferation via stimulating TNF-alpha release and H2O2 derived from NADPH oxidase: a cell culture study	scientific article

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scholarly article

1 reference

Europe PubMed Central

24 July 2017

Fibrillar beta-amyloid peptide Abeta1-40 activates microglial proliferation via stimulating TNF-alpha release and H2O2 derived from NADPH oxidase: a cell culture study (English)

1 reference

Europe PubMed Central

24 July 2017

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Guy C Brown

5

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author name string

Aiste Jekabsone

1

1 reference

Europe PubMed Central

24 July 2017

Palwinder K Mander

2

1 reference

Europe PubMed Central

24 July 2017

Anna Tickler

3

1 reference

Europe PubMed Central

24 July 2017

Martyn Sharpe

4

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Europe PubMed Central

24 July 2017

publication date

7 September 2006

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Europe PubMed Central

24 July 2017

Journal of Neuroinflammation

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Europe PubMed Central

24 July 2017

3

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Europe PubMed Central

24 July 2017

24

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Europe PubMed Central

24 July 2017

https://scigraph.springernature.com/pub.10.1186/1742-2094-3-24

0 references

Microglia proliferation is regulated by hydrogen peroxide from NADPH oxidase

1 reference

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Activation of microglial NADPH oxidase is synergistic with glial iNOS expression in inducing neuronal death: a dual-key mechanism of inflammatory neurodegeneration

1 reference

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Microglia and inflammation-mediated neurodegeneration: multiple triggers with a common mechanism

1 reference

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Inhibition of granulocyte-macrophage colony-stimulating factor signaling and microglial proliferation by anti-CD45RO: role of Hck tyrosine kinase and phosphatidylinositol 3-kinase/Akt.

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Redox regulation of glial inflammatory response to lipopolysaccharide and interferongamma

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Inflammatory processes in Alzheimer's disease

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A cell surface receptor complex for fibrillar beta-amyloid mediates microglial activation.

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beta-amyloid activates the O-2 forming NADPH oxidase in microglia, monocytes, and neutrophils. A possible inflammatory mechanism of neuronal damage in Alzheimer's disease.

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Thioflavine T interaction with synthetic Alzheimer's disease beta-amyloid peptides: detection of amyloid aggregation in solution

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Relative abundance of Alzheimer A beta amyloid peptide variants in Alzheimer disease and normal aging

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Relationship of microglia and astrocytes to amyloid deposits of Alzheimer disease.

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Turnover of resident microglia in the normal adult mouse brain

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Microglia enhance beta-amyloid peptide-induced toxicity in cortical and mesencephalic neurons by producing reactive oxygen species.

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NADPH oxidase activity is required for endothelial cell proliferation and migration

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Lectin staining of sheep microglia

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Identifiers

10.1186/1742-2094-3-24

1 reference

Europe PubMed Central

24 July 2017

Dimensions Publication ID

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https://scigraph.springernature.com/pub.10.1186/1742-2094-3-24

9 September 2019

1 reference

Europe PubMed Central

24 July 2017

1 reference

Europe PubMed Central

24 July 2017

ResearchGate publication ID

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