Modulation of microglial/macrophage activation by macrophage inhibitory factor (TKP) or tuftsin (TKPR) attenuates the disease course of experimental autoimmune encephalomyelitis (Q33290993)

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English	Modulation of microglial/macrophage activation by macrophage inhibitory factor (TKP) or tuftsin (TKPR) attenuates the disease course of experimental autoimmune encephalomyelitis	scientific article

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scholarly article

1 reference

Europe PubMed Central

25 July 2017

Modulation of microglial/macrophage activation by macrophage inhibitory factor (TKP) or tuftsin (TKPR) attenuates the disease course of experimental autoimmune encephalomyelitis (English)

1 reference

Europe PubMed Central

25 July 2017

0 references

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experimental autoimmune encephalomyelitis

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author name string

Madhuri Bhasin

1

1 reference

Europe PubMed Central

25 July 2017

Muzhou Wu

2

1 reference

Europe PubMed Central

25 July 2017

Stella E Tsirka

3

1 reference

Europe PubMed Central

25 July 2017

publication date

16 July 2007

1 reference

Europe PubMed Central

25 July 2017

1 reference

Europe PubMed Central

25 July 2017

8

1 reference

Europe PubMed Central

25 July 2017

10

1 reference

Europe PubMed Central

25 July 2017

https://scigraph.springernature.com/pub.10.1186/1471-2172-8-10

0 references

Tim-2 regulates T helper type 2 responses and autoimmunity

1 reference

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Is angiotensin-II an endogenous pro-inflammatory molecule?

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N-acetyl-L-cysteine ameliorates the inflammatory disease process in experimental autoimmune encephalomyelitis in Lewis rats

1 reference

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Tuftsin fragment 1-3 is beneficial when delivered after the induction of intracerebral hemorrhage.

1 reference

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Tuftsin-AZT conjugate: potential macrophage targeting for AIDS therapy.

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New insights into cell responses involved in experimental autoimmune encephalomyelitis and multiple sclerosis.

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Involvement of tissue plasminogen activator in onset and effector phases of experimental allergic encephalomyelitis

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Coordinated induction of extracellular proteolysis systems during experimental autoimmune encephalomyelitis in mice

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Treatment with anti-CR3 antibodies ED7 and ED8 suppresses experimental allergic encephalomyelitis in Lewis rats.

1 reference

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Isolation and subunit composition of tuftsin receptor

1 reference

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The characteristics of purified HL60 tuftsin receptors

1 reference

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An elevated matrix metalloproteinase (MMP) in an animal model of multiple sclerosis is protective by affecting Th1/Th2 polarization.

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Experimental autoimmune encephalomyelitis repressed by microglial paralysis.

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Beta blocker and angiotensin-converting enzyme inhibitor therapy is associated with decreased Th1/Th2 cytokine ratios and inflammatory cytokine production in patients with chronic heart failure

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Tissue plasminogen activator mediates microglial activation via its finger domain through annexin II.

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Regulation of microglial expression of integrins by poly(ADP-ribose) polymerase-1.

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B-cell-deficient mice develop experimental allergic encephalomyelitis with demyelination after myelin oligodendrocyte glycoprotein sensitization.

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Excitotoxin-induced neuronal degeneration and seizure are mediated by tissue plasminogen activator.

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Glucocorticoid amplifies IL-2-dependent expansion of functional FoxP3(+)CD4(+)CD25(+) T regulatory cells in vivo and enhances their capacity to suppress EAE

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10.1186/1471-2172-8-10

1 reference

Europe PubMed Central

25 July 2017

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1 reference

Europe PubMed Central

25 July 2017

1 reference

Europe PubMed Central

25 July 2017

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