Alternative polyadenylation signals and promoters act in concert to control tissue-specific expression of the Opitz Syndrome gene MID1. (Q33305715)

25 July 2017

Alternative polyadenylation signals and promoters act in concert to control tissue-specific expression of the Opitz Syndrome gene MID1. (English)

1 reference

25 July 2017

Jennifer Winter

1

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25 July 2017

Melanie Kunath

2

1 reference

25 July 2017

Stefan Roepcke

3

1 reference

25 July 2017

Sven Krause

4

1 reference

25 July 2017

Rainer Schneider

5

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25 July 2017

Susann Schweiger

6

1 reference

25 July 2017

15 November 2007

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25 July 2017

BMC Molecular Biology

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25 July 2017

8

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25 July 2017

105

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https://scigraph.springernature.com/pub.10.1186/1471-2199-8-105

25 July 2017

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Regulation of the MID1 protein function is fine-tuned by a complex pattern of alternative splicing

21 June 2018

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Duplication of the MID1 first exon in a patient with Opitz G/BBB syndrome

21 June 2018

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21 June 2018

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Widely spaced alternative promoters, conserved between human and rodent, control expression of the Opitz syndrome gene MID1

21 June 2018

1 reference

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21 June 2018

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21 June 2018

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21 June 2018

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MID1, mutated in Opitz syndrome, encodes an ubiquitin ligase that targets phosphatase 2A for degradation

21 June 2018

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A complex containing CstF-64 and the SL2 snRNP connects mRNA 3' end formation and trans-splicing in C. elegans operons.

21 June 2018

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21 June 2018

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21 June 2018

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21 June 2018

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The Opitz syndrome gene product, MID1, associates with microtubules

21 June 2018

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21 June 2018

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Opitz G/BBB syndrome, a defect of midline development, is due to mutations in a new RING finger gene on Xp22

21 June 2018

1 reference

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Further analysis of cytoplasmic polyadenylation in Xenopus embryos and identification of embryonic cytoplasmic polyadenylation element-binding proteins

21 June 2018

1 reference

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21 June 2018

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Embryonic expression of the human MID1 gene and its mutations in Opitz syndrome.

31 October 2018

1 reference

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Isolation and characterisation of the chick orthologue of the Opitz syndrome gene, Mid1, supports a conserved role in vertebrate development.

31 October 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2248598

31 October 2018

Identifiers

DOI

10.1186/1471-2199-8-105

1 reference

25 July 2017

1 reference

25 July 2017

1 reference