Palmitate-induced beta-cell dysfunction is associated with excessive NO production and is reversed by thiazolidinedione-mediated inhibition of GPR40 transduction mechanisms (Q33334424)
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English | Palmitate-induced beta-cell dysfunction is associated with excessive NO production and is reversed by thiazolidinedione-mediated inhibition of GPR40 transduction mechanisms |
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Palmitate-induced beta-cell dysfunction is associated with excessive NO production and is reversed by thiazolidinedione-mediated inhibition of GPR40 transduction mechanisms (English)
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Sandra Meidute Abaraviciene
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Ingmar Lundquist
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Juris Galvanovskis
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Erik Flodgren
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Björn Olde
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14 May 2008
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