Intracellular ATP, a switch in the decision between apoptosis and necrosis. (Q33533984)

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10 November 2019

review article

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title

Intracellular ATP, a switch in the decision between apoptosis and necrosis (English)

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10 November 2019

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2

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10 November 2019

author name string

P Nicotera

series ordinal

1

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10 November 2019

E Ferrando-May

series ordinal

3

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10 November 2019

publication date

1 December 1998

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10 November 2019

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10 November 2019

volume

102-103

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10 November 2019

page(s)

139-142

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Selective induction of apoptosis in Hep 3B cells by topoisomerase I inhibitors: evidence for a protease-dependent pathway that does not activate cysteine protease P32.

10 November 2019

cites work

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Induction of the mitochondrial permeability transition by protease activity in rats: a mechanism of hepatocyte necrosis

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Glutamate-induced neuronal death: a succession of necrosis or apoptosis depending on mitochondrial function

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Different subcellular distribution of caspase-3 and caspase-7 following Fas-induced apoptosis in mouse liver

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Proteasomes play an essential role in thymocyte apoptosis

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Mitochondrial calcium transport: physiological and pathological relevance

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The apoptosis-necrosis paradox. Apoptogenic proteases activated after mitochondrial permeability transition determine the mode of cell death.

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7 January 2021

Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics

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The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

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Altered cytokine export and apoptosis in mice deficient in interleukin-1 beta converting enzyme

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Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice

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Intracellular adenosine triphosphate (ATP) concentration: a switch in the decision between apoptosis and necrosis

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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7 January 2021

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

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S-nitrosylation regulates apoptosis.

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Target cell lysis by CTL granule exocytosis is independent of ICE/Ced-3 family proteases

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Programmed cell death, apoptosis and killer genes

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Do all programmed cell deaths occur via apoptosis?

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Developing Caenorhabditis elegans neurons may contain both cell-death protective and killer activities

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The central executioner of apoptosis: multiple connections between protease activation and mitochondria in Fas/APO-1/CD95- and ceramide-induced apoptosis

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Inhibition of caspases increases the sensitivity of L929 cells to necrosis mediated by tumor necrosis factor.

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Caspases and caspase inhibitors

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Constitutive expression of the machinery for programmed cell death

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BAX-induced cell death may not require interleukin 1 beta-converting enzyme-like proteases

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Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

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7 January 2021

Essential role of active nuclear transport in apoptosis

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The Caenorhabditis elegans genes ced-3 and ced-4 act cell autonomously to cause programmed cell death

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7 January 2021

Mitochondrial control of nuclear apoptosis.

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7 January 2021

10.1016/S0378-4274(98)00298-7

Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10022245%20AND%20SRC:MED&resulttype=core&format=json

10 November 2019

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