Hypomethylation of the CTCFL/BORIS promoter and aberrant expression during endometrial cancer progression suggests a role as an Epi-driver gene. (Q33572253)

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English	Hypomethylation of the CTCFL/BORIS promoter and aberrant expression during endometrial cancer progression suggests a role as an Epi-driver gene.	scientific article

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11 March 2020

Hypomethylation of the CTCFL/BORIS promoter and aberrant expression during endometrial cancer progression suggests a role as an Epi-driver gene (English)

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11 March 2020

endometrial cancer

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inferred from title

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11 March 2020

Camilla Krakstad

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11 March 2020

Martin Widschwendter

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Jone Trovik

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Helga B Salvesen

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11 March 2020

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Erling A Hoivik

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11 March 2020

Kanthida Kusonmano

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11 March 2020

Anna Berg

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11 March 2020

Elisabeth Wik

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11 March 2020

Henrica M J Werner

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11 March 2020

Kjell Petersen

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11 March 2020

Anne M Oyan

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11 March 2020

Karl-Henning Kalland

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1 February 2014

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11 March 2020

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11 March 2020

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11 March 2020

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11 March 2020

1052-1061

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11 March 2020

Role of DNA methylation and epigenetic silencing of HAND2 in endometrial cancer development

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High frequency strand slippage mutations in CTCF in MSI-positive endometrial cancers.

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De novo mutations in the genome organizer CTCF cause intellectual disability.

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BORIS, brother of the regulator of imprinted sites, is aberrantly expressed in hepatocellular carcinoma

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Exome sequencing of serous endometrial tumors identifies recurrent somatic mutations in chromatin-remodeling and ubiquitin ligase complex genes

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21 June 2018

Markers for individualised therapy in endometrial carcinoma

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21 June 2018

Cancer epigenetics: from mechanism to therapy

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21 June 2018

The male germ cell gene regulator CTCFL is functionally different from CTCF and binds CTCF-like consensus sites in a nucleosome composition-dependent manner

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CTCF: insights into insulator function during development

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BORIS in human cancers -- a review

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The role of androgens and the androgen receptor in cycling endometrium

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Chromatin insulators: a role in nuclear organization and gene expression

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Cancer-testis antigen, BORIS based vaccine delivered by dendritic cells is extremely effective against a very aggressive and highly metastatic mouse mammary carcinoma.

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The tumor suppressor role of CTCF.

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Cancer epigenetics reaches mainstream oncology

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Tumor regression in patients with metastatic synovial cell sarcoma and melanoma using genetically engineered lymphocytes reactive with NY-ESO-1

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The structural complexity of the human BORIS gene in gametogenesis and cancer

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BORIS/CTCFL-mediated transcriptional regulation of the hTERT telomerase gene in testicular and ovarian tumor cells

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DNA methylation in endometrial cancer

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21 June 2018

Does CTCF mediate between nuclear organization and gene expression?

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21 June 2018

Expression of the CTCF-paralogous cancer-testis gene, brother of the regulator of imprinted sites (BORIS), is regulated by three alternative promoters modulated by CpG methylation and by CTCF and p53 transcription factors

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Reciprocal binding of CTCF and BORIS to the NY-ESO-1 promoter coincides with derepression of this cancer-testis gene in lung cancer cells

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Conditional expression of the CTCF-paralogous transcriptional factor BORIS in normal cells results in demethylation and derepression of MAGE-A1 and reactivation of other cancer-testis genes

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Lack of estrogen receptor-α is associated with epithelial-mesenchymal transition and PI3K alterations in endometrial carcinoma

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Endometrial Carcinoma

1 reference

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10.18632/ONCOTARGET.1697

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