Procoagulant and anticoagulant properties of coagulation factor V: factor V Leiden (APC resistance) causes hypercoagulability by dual mechanisms (Q33611607)

From Wikidata

Jump to navigation Jump to search

scientific article

Language	Label	Description	Also known as
English	Procoagulant and anticoagulant properties of coagulation factor V: factor V Leiden (APC resistance) causes hypercoagulability by dual mechanisms	scientific article

Statements

scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

28 July 2017

1 reference

Europe PubMed Central

Procoagulant and anticoagulant properties of coagulation factor V: factor V Leiden (APC resistance) causes hypercoagulability by dual mechanisms (English)

1 reference

Europe PubMed Central

PubMed publication ID

28 July 2017

anticoagulation

1 reference

based on heuristic

inferred from title

1 reference

based on heuristic

inferred from title

1 reference

based on heuristic

inferred from title

Factor V Leiden

1 reference

based on heuristic

inferred from title

author name string

Dahlbäck B

1

1 reference

Europe PubMed Central

PubMed publication ID

28 July 2017

publication date

1 May 1999

1 reference

Europe PubMed Central

PubMed publication ID

28 July 2017

Translational Research

1 reference

Europe PubMed Central

PubMed publication ID

28 July 2017

133

1 reference

Europe PubMed Central

PubMed publication ID

28 July 2017

5

1 reference

Europe PubMed Central

PubMed publication ID

28 July 2017

415-422

1 reference

Europe PubMed Central

PubMed publication ID

28 July 2017

The protein C anticoagulant system: inherited defects as basis for venous thrombosis

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Inherited resistance to activated protein C is corrected by anticoagulant cofactor activity found to be a property of factor V.

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Thrombomodulin as a model of molecular mechanisms that modulate protease specificity and function at the vessel surface

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Cleavage requirements for activation of factor V by factor Xa

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Activation of human factor V by factor Xa and thrombin

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Structural investigation of the A domains of human blood coagulation factor V by molecular modeling

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Peptide bond cleavages and loss of functional activity during inactivation of factor Va and factor VaR506Q by activated protein C.

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Effects of protein S and factor Xa on peptide bond cleavages during inactivation of factor Va and factor VaR506Q by activated protein C

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Protein S alters the active site location of activated protein C above the membrane surface. A fluorescence resonance energy transfer study of topography

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

The C-terminal region of the factor V B-domain is crucial for the anticoagulant activity of factor V.

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Familial thrombophilia due to a previously unrecognized mechanism characterized by poor anticoagulant response to activated protein C: prediction of a cofactor to activated protein C

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Mutation in blood coagulation factor V associated with resistance to activated protein C.

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Activated protein C resistance caused by Arg506Gln mutation in factor Va

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Association of idiopathic venous thromboembolism with single point-mutation at Arg506 of factor V

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Linkage between inherited resistance to activated protein C and factor V gene mutation in venous thrombosis

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Characterization of the molecular defect in factor VR506Q

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Molecular mechanisms of activated protein C resistance. Properties of factor V isolated from an individual with homozygosity for the Arg506 to Gln mutation in the factor V gene

1 reference

https://api.crossref.org/works/10.1016%2FS0022-2143%2899%2990018-5

7 January 2021

Identifiers

10.1016/S0022-2143(99)90018-5

1 reference

Europe PubMed Central

PubMed publication ID

28 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

28 July 2017

ResearchGate publication ID

0 references

Sitelinks

Wikipedia(0 entries)

Wikibooks(0 entries)

Wikinews(0 entries)

Wikiquote(0 entries)

Wikisource(0 entries)

Wikiversity(0 entries)

Wikivoyage(0 entries)

Wiktionary(0 entries)

Multilingual sites(0 entries)

Retrieved from "https://www.wikidata.org/w/index.php?title=Q33611607&oldid=1542307145"