Parasite-derived plasma microparticles contribute significantly to malaria infection-induced inflammation through potent macrophage stimulation (Q33618034)

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English	Parasite-derived plasma microparticles contribute significantly to malaria infection-induced inflammation through potent macrophage stimulation	scientific article

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17 January 2020

Parasite-derived plasma microparticles contribute significantly to malaria infection-induced inflammation through potent macrophage stimulation (English)

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17 January 2020

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Kevin N. Couper

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Eleanor M Riley

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Tom Barnes

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Julius C R Hafalla

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Thomas Secher

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17 January 2020

J Brian de Souza

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Bernhard Ryffel

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29 January 2010

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17 January 2020

e1000744

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17 January 2020

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29 January 2010

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MAPK-activated protein kinase 2 differentially regulates plasmodium falciparum glycosylphosphatidylinositol-induced production of tumor necrosis factor-{alpha} and interleukin-12 in macrophages

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MyD88/IL-18-dependent pathways rather than TLRs control early parasitaemia in non-lethal Plasmodium yoelii infection

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Malaria impairs T cell clustering and immune priming despite normal signal 1 from dendritic cells

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Macrophage-mediated but gamma interferon-independent innate immune responses control the primary wave of Plasmodium yoelii parasitemia

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Murine cerebral malaria development is independent of toll-like receptor signaling

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21 June 2018

Malaria hemozoin is immunologically inert but radically enhances innate responses by presenting malaria DNA to Toll-like receptor 9.

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Acquisition of hemozoin by monocytes down-regulates interleukin-12 p40 (IL-12p40) transcripts and circulating IL-12p70 through an IL-10-dependent mechanism: in vivo and in vitro findings in severe malarial anemia

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Cell vesiculation and immunopathology: implications in cerebral malaria

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Suppression of adaptive immunity to heterologous antigens during Plasmodium infection through hemozoin-induced failure of dendritic cell function

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21 June 2018

Cerebral malaria: role of microparticles and platelets in alterations of the blood-brain barrier

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21 June 2018

Toll-like receptor (TLR) polymorphisms in African children: Common TLR-4 variants predispose to severe malaria

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21 June 2018

Platelets potentiate brain endothelial alterations induced by Plasmodium falciparum

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21 June 2018

Natural killer cell and macrophage cooperation in MyD88-dependent innate responses to Plasmodium falciparum

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21 June 2018

Selected Toll-like receptor agonist combinations synergistically trigger a T helper type 1-polarizing program in dendritic cells

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21 June 2018

Development and regulation of cell-mediated immune responses to the blood stages of malaria: implications for vaccine research

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21 June 2018

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21 June 2018

ABCA1 gene deletion protects against cerebral malaria: potential pathogenic role of microparticles in neuropathology.

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21 June 2018

Toll-like receptor 9 mediates innate immune activation by the malaria pigment hemozoin

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21 June 2018

Induction of proinflammatory responses in macrophages by the glycosylphosphatidylinositols of Plasmodium falciparum: cell signaling receptors, glycosylphosphatidylinositol (GPI) structural requirement, and regulation of GPI activity

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21 June 2018

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21 June 2018

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Brain macrophage activation in murine cerebral malaria precedes accumulation of leukocytes and CD8+ T cell proliferation.

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Plasmodium berghei infection in mice induces liver injury by an IL-12- and toll-like receptor/myeloid differentiation factor 88-dependent mechanism.

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21 January 2018

Pathological role of Toll-like receptor signaling in cerebral malaria.

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Toll-like receptor modulation of murine cerebral malaria is dependent on the genetic background of the host.

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21 January 2018

Disruption of CD36 impairs cytokine response to Plasmodium falciparum glycosylphosphatidylinositol and confers susceptibility to severe and fatal malaria in vivo.

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Killing of the malarial parasite Plasmodium yoelii in vitro by cells of myeloid origin

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21 January 2018

Induction of proinflammatory responses in macrophages by the glycosylphosphatidylinositols of Plasmodium falciparum: the requirement of extracellular signal-regulated kinase, p38, c-Jun N-terminal kinase and NF-kappaB pathways for the expression of

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Phagocytosis of Leishmania mexicana amastigotes by macrophages leads to a sustained suppression of IL-12 production

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inferred from PubMed ID database lookup

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10.1371/JOURNAL.PPAT.1000744

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17 January 2020

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17 January 2020

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