Distinct lipid a moieties contribute to pathogen-induced site-specific vascular inflammation (Q33882825)

25010102

29 July 2017

title

Distinct lipid a moieties contribute to pathogen-induced site-specific vascular inflammation (English)

1 reference

29 July 2017

0 references

8

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11 June 2020

author name string

Connie Slocum

1

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11 June 2020

Stephen R Coats

2

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11 June 2020

Ning Hua

3

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11 June 2020

Carolyn Kramer

4

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11 June 2020

George Papadopoulos

5

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11 June 2020

Ellen O Weinberg

6

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11 June 2020

Cynthia V Gudino

7

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11 June 2020

Richard P Darveau

9

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11 June 2020

Caroline A Genco

10

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11 June 2020

publication date

10 July 2014

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29 July 2017

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11 June 2020

volume

10

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11 June 2020

issue

7

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11 June 2020

page(s)

e1004215

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Creative Commons Attribution 4.0 International

11 June 2020

copyright license

start time

10 July 2014

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April 2022 Public Data File from Crossref

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copyrighted

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describes a project that uses

ImageJ

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11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC4092147/fullTextXML

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

1 reference

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5 July 2018

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5 July 2018

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5 July 2018

1 reference

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5 July 2018

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Porphyromonas gingivalis lipopolysaccharide lipid A heterogeneity: differential activities of tetra- and penta-acylated lipid A structures on E-selectin expression and TLR4 recognition.

5 July 2018

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Characterization of late acyltransferase genes of Yersinia pestis and their role in temperature-dependent lipid A variation

5 July 2018

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5 July 2018

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MD-2 mediates the ability of tetra-acylated and penta-acylated lipopolysaccharides to antagonize Escherichia coli lipopolysaccharide at the TLR4 signaling complex

5 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4092147

Porphyromonas gingivalis fimbria-dependent activation of inflammatory genes in human aortic endothelial cells.

5 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4092147

Porphyromonas gingivalis lipopolysaccharide displays functionally diverse interactions with the innate host defense system

5 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4092147

LPS, TLR4 and infectious disease diversity.

5 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4092147

Lack of Toll-like receptor 4 or myeloid differentiation factor 88 reduces atherosclerosis and alters plaque phenotype in mice deficient in apolipoprotein E

5 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4092147

NALPs: a novel protein family involved in inflammation

5 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4092147

Modification of the structure and activity of lipid A in Yersinia pestis lipopolysaccharide by growth temperature

5 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4092147

Lipopolysaccharide endotoxins

5 July 2018

1 reference

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5 July 2018

1 reference

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Identification of periodontal pathogens in atheromatous plaques.

5 July 2018

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5 July 2018

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Periodontal diseases in the United States population

5 July 2018

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Structural study on the free lipid A isolated from lipopolysaccharide of Porphyromonas gingivalis

5 July 2018

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5 July 2018

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Human Atherosclerotic Plaque Contains Viable Invasive Actinobacillus actinomycetemcomitans and Porphyromonas gingivalis

21 January 2018

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Innate immune recognition of invasive bacteria accelerates atherosclerosis in apolipoprotein E-deficient mice

21 January 2018

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Pathogen-mediated inflammatory atherosclerosis is mediated in part via Toll-like receptor 2-induced inflammatory responses.

21 January 2018

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Fimbrial proteins of porphyromonas gingivalis mediate in vivo virulence and exploit TLR2 and complement receptor 3 to persist in macrophages.

21 January 2018

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21 January 2018

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12 December 2020

Fimbria-dependent activation of pro-inflammatory molecules in Porphyromonas gingivalis infected human aortic endothelial cells

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Porphyromonas gingivalis antagonises Campylobacter rectus induced cytokine production by human monocytes

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High Antibody Levels to P. gingivalis in Cardiovascular Disease

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DOI

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