Placenta growth factor (PlGF), a novel inducer of plasminogen activator inhibitor-1 (PAI-1) in sickle cell disease (SCD). (Q33883207)

29 July 2017

title

Placenta growth factor (PlGF), a novel inducer of plasminogen activator inhibitor-1 (PAI-1) in sickle cell disease (SCD). (English)

1 reference

29 July 2017

0 references

1 reference

Nitin Patel

1

1 reference

29 July 2017

Nambirajan Sundaram

2

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29 July 2017

Mingyan Yang

3

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29 July 2017

Catherine Madigan

4

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29 July 2017

Vijay K Kalra

5

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29 July 2017

Punam Malik

6

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29 July 2017

language of work or name

English

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publication date

29 March 2010

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Journal of Biological Chemistry

29 July 2017

published in

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29 July 2017

volume

285

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29 July 2017

issue

22

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29 July 2017

page(s)

16713-16722

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Placenta growth factor induces 5-lipoxygenase-activating protein to increase leukotriene formation in sickle cell disease

29 July 2017

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5 July 2018

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Oxidative stress activates the plasminogen activator inhibitor type 1 (PAI-1) promoter through an AP-1 response element and cooperates with insulin for additive effects on PAI-1 transcription

5 July 2018

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5 July 2018

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Identification of a tightly regulated hypoxia-response element in the promoter of human plasminogen activator inhibitor-1.

5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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Pulmonary hypertension as a risk factor for death in patients with sickle cell disease

5 July 2018

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Pathogenesis and treatment of sickle cell disease

21 January 2018

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Coagulation activation and inflammation in sickle cell disease-associated pulmonary hypertension.

21 January 2018

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Absence of host plasminogen activator inhibitor 1 prevents cancer invasion and vascularization.

21 January 2018

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Induction of the plasminogen activator inhibitor-1 gene expression by mild hypoxia via a hypoxia response element binding the hypoxia-inducible factor-1 in rat hepatocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M110.101691

Placenta growth factor activates monocytes and correlates with sickle cell disease severity

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M110.101691

Transgenic knockout mice with exclusively human sickle hemoglobin and sickle cell disease

21 January 2018

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Correction of a mouse model of sickle cell disease: lentiviral/antisickling beta-globin gene transduction of unmobilized, purified hematopoietic stem cells

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M110.101691

Synergism between vascular endothelial growth factor and placental growth factor contributes to angiogenesis and plasma extravasation in pathological conditions.

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M110.101691

Hypoxia-inducible factor-1 and hypoxia response elements mediate the induction of plasminogen activator inhibitor-1 gene expression by insulin in primary rat hepatocytes

21 January 2018

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12 December 2020

Enhanced platelet reactivity and hypercoagulability in the steady state of sickle cell anaemia

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12 December 2020

Abnormalities of coagulation and fibrinolysis in homozygous sickle cell disease

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12 December 2020

Whole blood tissue factor procoagulant activity is elevated in patients with sickle cell disease

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12 December 2020

Tumor necrosis factor-alpha-induced release of plasminogen activator inhibitor-1 from human umbilical vein endothelial cells: involvement of intracellular ceramide signaling event

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12 December 2020

Antiphospholipid antibodies, proteins C and S, and coagulation changes in sickle cell disease

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Thrombogenesis in sickle cell disease

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12 December 2020

Identifiers

DOI

10.1074/JBC.M110.101691

1 reference

29 July 2017

1 reference

29 July 2017

1 reference