Reversing neurodegeneration: a promise unfolds (Q33897927)

29 July 2017

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title

Reversing neurodegeneration: a promise unfolds (English)

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29 July 2017

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2

H Y Zoghbi

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29 July 2017

H T Orr

1

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29 July 2017

language of work or name

English

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publication date

1 March 2000

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29 July 2017

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29 July 2017

volume

101

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29 July 2017

issue

1

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29 July 2017

page(s)

1-4

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Ubiquitin, cellular inclusions and their role in neurodegeneration

29 July 2017

cites work

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Up-regulation of protein chaperones preserves viability of cells expressing toxic Cu/Zn-superoxide dismutase mutants associated with amyotrophic lateral sclerosis

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7 January 2021

Analysis of the role of heat shock protein (Hsp) molecular chaperones in polyglutamine disease

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Acceleration of oligomerization, not fibrillization, is a shared property of both alpha-synuclein mutations linked to early-onset Parkinson's disease: implications for pathogenesis and therapy

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Chaperone suppression of aggregation and altered subcellular proteasome localization imply protein misfolding in SCA1.

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Mutation of the E6-AP ubiquitin ligase reduces nuclear inclusion frequency while accelerating polyglutamine-induced pathology in SCA1 mice.

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A YAC mouse model for Huntington's disease with full-length mutant huntingtin, cytoplasmic toxicity, and selective striatal neurodegeneration.

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The tyrosine kinase negative regulator c-Cbl as a RING-type, E2-dependent ubiquitin-protein ligase

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Aberrant protein deposition and neurological disease

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Ataxin-1 nuclear localization and aggregation: role in polyglutamine-induced disease in SCA1 transgenic mice

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Mutations in the parkin gene cause autosomal recessive juvenile parkinsonism

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The ubiquitin pathway in Parkinson's disease

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7 January 2021

Expanding our understanding of polyglutamine diseases through mouse models

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7 January 2021

RING fingers mediate ubiquitin-conjugating enzyme (E2)-dependent ubiquitination

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Intragenic deletion in the gene encoding ubiquitin carboxy-terminal hydrolase in gad mice

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Suppression of polyglutamine-mediated neurodegeneration in Drosophila by the molecular chaperone HSP70.

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Reversal of neuropathology and motor dysfunction in a conditional model of Huntington's disease

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7 January 2021

10.1016/S0092-8674(00)80617-0

Identifiers

DOI

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29 July 2017

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