Dose-response curve and optimal dosing regimen of cyclosporin A after traumatic brain injury in rats (Q33924997)

30 July 2017

Dose-response curve and optimal dosing regimen of cyclosporin A after traumatic brain injury in rats (English)

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30 July 2017

traumatic brain injury

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Sullivan PG

1

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30 July 2017

Rabchevsky AG

2

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30 July 2017

Hicks RR

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30 July 2017

Gibson TR

4

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30 July 2017

Fletcher-Turner A

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30 July 2017

Scheff SW

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30 July 2017

language of work or name

English

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publication date

1 January 2000

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30 July 2017

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30 July 2017

101

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30 July 2017

2

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30 July 2017

289-295

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Do NMDA antagonists prevent neuronal injury? Yes

30 July 2017

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Glutamate-induced neuronal death: A succession of necrosis or apoptosis depending on mitochondrial function

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Blood-brain barrier breach following cortical contusion in the rat.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Neuronal cell loss in the CA3 subfield of the hippocampus following cortical contusion utilizing the optical disector method for cell counting.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

The biphasic opening of the blood-brain barrier in the cortex and hippocampus after traumatic brain injury in rats.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Post-ischemic administration of HU-211, a novel non-competitive NMDA antagonist, protects against blood-brain barrier disruption in photochemical cortical infarction in rats: a quantitative study.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Severe central-nervous-system toxicity associated with cyclosporin.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

The permeability transition pore as a mitochondrial calcium release channel: a critical appraisal

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Persistent metabolic sequelae of severe head injury in humans in vivo.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Excitotoxic cell death

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Oxidative stress, glutamate, and neurodegenerative disorders

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Central nervous system toxicity after liver transplantation. The role of cyclosporine and cholesterol.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Ruthenium red protects against glutamate-induced neuronal death in cerebellar culture

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Early microvascular and neuronal consequences of traumatic brain injury: a light and electron microscopic study in rats.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Dextrorphan reduces infarct volume, vascular injury, and brain edema after ischemic brain injury.

1 reference

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7 January 2021

Mitochondrial production of reactive oxygen species in cortical neurons following exposure to N-methyl-D-aspartate

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

The role of excitatory amino acids and NMDA receptors in traumatic brain injury

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Central nervous system toxicity of cyclosporine in a rat model

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Changes in the bioenergetic state of rat hippocampus during 2.5 min of ischemia, and prevention of cell damage by cyclosporin A in hyperglycemic subjects.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Cyclosporin A, but not FK 506, protects mitochondria and neurons against hypoglycemic damage and implicates the mitochondrial permeability transition in cell death.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Serum extravasation and cytoskeletal alterations following traumatic brain injury in rats. Comparison of lateral fluid percussion and cortical impact models.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

1 reference

7 January 2021

Effect of cyclosporin A or tacrolimus on the function of blood-brain barrier cells.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Capillary NMDA receptors regulate blood-brain barrier function and breakdown

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Mitochondrial permeability transition in the central nervous system: induction by calcium cycling-dependent and -independent pathways.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Amelioration by cyclosporin A of brain damage following 5 or 10 min of ischemia in rats subjected to preischemic hyperglycemia.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Calcium, free radicals, and excitotoxic neuronal death in primary cell culture

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Endogenous neuroprotection factors and traumatic brain injury: mechanisms of action and implications for therapy

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Novel pharmacologic therapies in the treatment of experimental traumatic brain injury: a review

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Novel pharmacologic strategies in the treatment of experimental traumatic brain injury: 1998.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Traumatic brain injury in the rat: characterization of a lateral fluid-percussion model.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

NMDA- and endothelin-1-induced increases in blood-brain barrier permeability quantitated with Lucifer yellow.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Vascular changes in the spinal cord in N-methyl-D-aspartate-induced excitotoxicity: morphological and permeability studies

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Mitochondria and neuronal glutamate excitotoxicity.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Cyclosporin A limits calcium-induced axonal damage following traumatic brain injury

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

An intrathecal bolus of cyclosporin A before injury preserves mitochondrial integrity and attenuates axonal disruption in traumatic brain injury

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Pretreatment with NMDA antagonists limits release of excitatory amino acids following traumatic brain injury

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Workshop on animal models of traumatic brain injury

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Mitochondrial transmembrane potential and free radical production in excitotoxic neurodegeneration

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Failure of autoregulation after closed head injury: an experimental model.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Effects of oxidants and glutamate receptor activation on mitochondrial membrane potential in rat forebrain neurons.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Morris water maze deficits in rats following traumatic brain injury: lateral controlled cortical impact.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Cyclosporin A significantly ameliorates cortical damage following experimental traumatic brain injury in rodents.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

NMDA-induced superoxide production and neurotoxicity in cultured rat hippocampal neurons: role of mitochondria.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Cyclosporin A protects against ischemia-reperfusion injury in the brain

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Mechanisms of secondary brain damage in global and focal ischemia: a speculative synthesis

1 reference

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7 January 2021

Altered mitochondrial respiration in selectively vulnerable brain subregions following transient forebrain ischemia in the rat.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Systematic studies on the effects of the NMDA receptor antagonist MK-801 on cerebral blood flow and responsivity, EEG, and blood-brain barrier following complete reversible cerebral ischemia.

1 reference

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7 January 2021

Glutamate-induced neuron death requires mitochondrial calcium uptake

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Exacerbation of damage and altered NF-kappaB activation in mice lacking tumor necrosis factor receptors after traumatic brain injury.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Traumatic brain injury alters synaptic homeostasis: implications for impaired mitochondrial and transport function.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Continuous infusion of cyclosporin A postinjury significantly ameliorates cortical damage following traumatic brain injury

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Cyclosporin A attenuates acute mitochondrial dysfunction following traumatic brain injury.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Glutamate-induced calcium loads: effects on energy metabolism and neuronal viability

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Amelioration by cyclosporin A of brain damage in transient forebrain ischemia in the rat.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Cyclosporin A dramatically ameliorates CA1 hippocampal damage following transient forebrain ischaemia in the rat.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Mitochondrial dysfunction after experimental and human brain injury and its possible reversal with a selective N-type calcium channel antagonist (SNX-111).

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Sequestration of glutamate-induced Ca2+ loads by mitochondria in cultured rat hippocampal neurons

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Mitochondrial depolarization in glutamate-stimulated neurons: an early signal specific to excitotoxin exposure

1 reference

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7 January 2021

Mitochondria accumulate Ca2+ following intense glutamate stimulation of cultured rat forebrain neurones.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

Mitochondrial dysfunction and calcium perturbation induced by traumatic brain injury.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

The calcium content of mitochondria from brain subregions following short-term forebrain ischemia and recirculation in the rat.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2800%2900380-8

7 January 2021

10.1016/S0306-4522(00)00380-8

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DOI

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release_tg673ppstzhg7m64ozszxltqla

30 July 2017

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https://api.fatcat.wiki/v0/release/tg673ppstzhg7m64ozszxltqla

24 November 2022

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