Hyaluronan signaling during ozone-induced lung injury requires TLR4, MyD88, and TIRAP. (Q34071823)

30 July 2017

Hyaluronan signaling during ozone-induced lung injury requires TLR4, MyD88, and TIRAP. (English)

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30 July 2017

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1 reference

Stavros Garantziotis

3

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author name string

Zhuowei Li

1

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30 July 2017

Erin N Potts-Kant

2

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30 July 2017

W Michael Foster

4

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30 July 2017

John W Hollingsworth

5

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30 July 2017

language of work or name

English

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publication date

4 November 2011

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30 July 2017

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30 July 2017

6

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30 July 2017

11

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30 July 2017

e27137

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Creative Commons CC0 License

30 July 2017

start time

4 November 2011

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April 2022 Public Data File from Crossref

copyright status

copyrighted, dedicated to the public domain by copyright holder

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cites work

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Essential role for TIRAP in activation of the signalling cascade shared by TLR2 and TLR4.

28 July 2018

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28 July 2018

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28 July 2018

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Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function

28 July 2018

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Hyaluronan (HA) fragments induce chemokine gene expression in alveolar macrophages. The role of HA size and CD44.

28 July 2018

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28 July 2018

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28 July 2018

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28 July 2018

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28 July 2018

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28 October 2018

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28 October 2018

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TLR4- and TLR2-mediated B cell responses control the clearance of the bacterial pathogen, Leptospira interrogans

28 October 2018

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Hyaluronan mediates ozone-induced airway hyperresponsiveness in mice.

28 October 2018

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Interleukin (IL)-1 regulates ozone-enhanced tracheal smooth muscle responsiveness by increasing substance P (SP) production in intrinsic airway neurons of ferret

28 October 2018

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IL-1R1/MyD88 signaling and the inflammasome are essential in pulmonary inflammation and fibrosis in mice

28 October 2018

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Recognition of hyaluronan released in sterile injury involves a unique receptor complex dependent on Toll-like receptor 4, CD44, and MD-2.

28 October 2018

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The MyD88-dependent, but not the MyD88-independent, pathway of TLR4 signaling is important in clearing nontypeable haemophilus influenzae from the mouse lung.

28 October 2018

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Cutting edge: TLR4 deficiency confers susceptibility to lethal oxidant lung injury.

28 October 2018

1 reference

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Ozone and short-term mortality in 95 US urban communities, 1987-2000.

28 October 2018

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The role of Toll-like receptor 4 in environmental airway injury in mice.

28 October 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3208559

Hyaluronan fragments stimulate endothelial recognition of injury through TLR4.

28 October 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3208559

Interleukin-1 receptor antagonist attenuates airway hyperresponsiveness following exposure to ozone

28 October 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3208559

Association of low-level ozone and fine particles with respiratory symptoms in children with asthma.

28 October 2018

1 reference

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Hyperresponsive airways correlate with lung tissue inflammatory cell changes in ozone-exposed rats

28 October 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3208559

TLR4 through IFN-β promotes low molecular mass hyaluronan-induced neutrophil apoptosis

28 October 2018

1 reference

12 December 2020

Genetic susceptibility to ozone-induced lung hyperpermeability: role of toll-like receptor 4

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12 December 2020

Role of TLR2, TLR4, and MyD88 in murine ozone-induced airway hyperresponsiveness and neutrophilia

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12 December 2020

Linkage analysis of susceptibility to ozone-induced lung inflammation in inbred mice

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12 December 2020

Role of neutrophil elastase in ozone-induced airway responses in guinea-pigs

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12 December 2020

IL-1 receptor-mediated signal is an essential component of MyD88-dependent innate response to Mycobacterium tuberculosis infection

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12 December 2020

10.1371/JOURNAL.PONE.0027137

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DOI

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30 July 2017

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30 July 2017

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