Modulating activity of vancomycin and daptomycin on the expression of autolysis cell-wall turnover and membrane charge genes in hVISA and VISA strains (Q34130471)

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English	Modulating activity of vancomycin and daptomycin on the expression of autolysis cell-wall turnover and membrane charge genes in hVISA and VISA strains	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

31 July 2017

Modulating activity of vancomycin and daptomycin on the expression of autolysis cell-wall turnover and membrane charge genes in hVISA and VISA strains (English)

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Europe PubMed Central

PubMed publication ID

31 July 2017

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based on heuristic

inferred from title

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inferred from title

Simona Purrello

object named as

Simona Purrello

4

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Cinzia Di Pietro

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Cinzia Di Pietro

6

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Stefania Stefani

object named as

Stefania Stefani

8

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1

object named as

Viviana Cafiso

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Europe PubMed Central

PubMed publication ID

31 July 2017

author name string

Taschia Bertuccio

2

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Europe PubMed Central

PubMed publication ID

31 July 2017

Daniela Spina

3

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Europe PubMed Central

PubMed publication ID

31 July 2017

Floriana Campanile

5

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Europe PubMed Central

PubMed publication ID

31 July 2017

Michele Purrello

7

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Europe PubMed Central

PubMed publication ID

31 July 2017

language of work or name

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publication date

9 January 2012

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Europe PubMed Central

PubMed publication ID

31 July 2017

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Europe PubMed Central

PubMed publication ID

31 July 2017

7

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Europe PubMed Central

PubMed publication ID

31 July 2017

1

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

e29573

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Europe PubMed Central

PubMed publication ID

31 July 2017

copyright license

Creative Commons Attribution 4.0 International

9 January 2012

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April 2022 Public Data File from Crossref

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walK and clpP mutations confer reduced vancomycin susceptibility in Staphylococcus aureus.

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Peptidoglycan crosslinking relaxation plays an important role in Staphylococcus aureus WalKR-dependent cell viability

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Broad-spectrum antimicrobial peptide resistance by MprF-mediated aminoacylation and flipping of phospholipids

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Regulation of mprF by antisense RNA restores daptomycin susceptibility to daptomycin-resistant isolates of Staphylococcus aureus

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Reduced vancomycin susceptibility in Staphylococcus aureus, including vancomycin-intermediate and heterogeneous vancomycin-intermediate strains: resistance mechanisms, laboratory detection, and clinical implications

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Enhanced expression of dltABCD is associated with the development of daptomycin nonsusceptibility in a clinical endocarditis isolate of Staphylococcus aureus

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New insights into the WalK/WalR (YycG/YycF) essential signal transduction pathway reveal a major role in controlling cell wall metabolism and biofilm formation in Staphylococcus aureus

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Interaction of the GraRS two-component system with the VraFG ABC transporter to support vancomycin-intermediate resistance in Staphylococcus aureus

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Autolytic properties of glycopeptide-intermediate Staphylococcus aureus Mu50.

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DltABCD- and MprF-mediated cell envelope modifications of Staphylococcus aureus confer resistance to platelet microbicidal proteins and contribute to virulence in a rabbit endocarditis model.

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The multifunctional Staphylococcus aureus autolysin aaa mediates adherence to immobilized fibrinogen and fibronectin

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Alterations of cell wall structure and metabolism accompany reduced susceptibility to vancomycin in an isogenic series of clinical isolates of Staphylococcus aureus

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Correlation of daptomycin bactericidal activity and membrane depolarization in Staphylococcus aureus

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Accessory gene regulator (agr) locus in geographically diverse Staphylococcus aureus isolates with reduced susceptibility to vancomycin

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The D-alanine residues of Staphylococcus aureus teichoic acids alter the susceptibility to vancomycin and the activity of autolytic enzymes

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10.1371/JOURNAL.PONE.0029573

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

2012PLoSO...729573C

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PMC publication ID

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Europe PubMed Central

PubMed publication ID

31 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

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