Transcriptional activity among high and low risk human papillomavirus E2 proteins correlates with E2 DNA binding (Q34150137)

31 July 2017

Transcriptional activity among high and low risk human papillomavirus E2 proteins correlates with E2 DNA binding (English)

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31 July 2017

Samuel Y Hou

1

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31 July 2017

Shwu-Yuan Wu

series ordinal

2

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31 July 2017

Cheng-Ming Chiang

series ordinal

3

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31 July 2017

language of work or name

English

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publication date

17 September 2002

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Journal of Biological Chemistry

31 July 2017

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31 July 2017

277

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31 July 2017

47

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31 July 2017

45619-45629

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Papillomaviruses causing cancer: evasion from host-cell control in early events in carcinogenesis

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TATA-binding protein-associated factors enhance the recruitment of RNA polymerase II by transcriptional activators

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Direct interaction between Sp1 and the BPV enhancer E2 protein mediates synergistic activation of transcription

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Functional interaction of a novel cellular protein with the papillomavirus E2 transactivation domain

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A Functional interaction between the human papillomavirus 16 transcription/replication factor E2 and the DNA damage response protein TopBP1

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Synergistic transcriptional-activation by the papillomavirus E2 protein occurs after DNA binding and correlates with a change in chromatin structure

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AMF-1/Gps2 binds p300 and enhances its interaction with papillomavirus E2 proteins

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cAMP response element-binding protein-binding protein binds to human papillomavirus E2 protein and activates E2-dependent transcription

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Functional interaction between p/CAF and human papillomavirus E2 protein

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The functional BPV-1 E2 trans-activating protein can act as a repressor by preventing formation of the initiation complex

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Alleviation of human papillomavirus E2-mediated transcriptional repression via formation of a TATA binding protein (or TFIID)-TFIIB-RNA polymerase II-TFIIF preinitiation complex

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A new cellular factor recognizes E2 binding sites of papillomaviruses which mediate transcriptional repression by E2.

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Human TATA binding protein inhibits human papillomavirus type 11 DNA replication by antagonizing E1-E2 protein complex formation on the viral origin of replication

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The E8E2C protein, a negative regulator of viral transcription and replication, is required for extrachromosomal maintenance of human papillomavirus type 31 in keratinocytes

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The E8 domain confers a novel long-distance transcriptional repression activity on the E8E2C protein of high-risk human papillomavirus type 31.

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Targeting the E1 replication protein to the papillomavirus origin of replication by complex formation with the E2 transactivator

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Binding of the human papillomavirus E1 origin-recognition protein is regulated through complex formation with the E2 enhancer-binding protein

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Segregation of viral plasmids depends on tethering to chromosomes and is regulated by phosphorylation

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E1 protein of bovine papillomavirus type 1 interferes with E2 protein-mediated tethering of the viral DNA to mitotic chromosomes

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Expression of the papillomavirus E2 protein in HeLa cells leads to apoptosis

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Abrogation of a mitotic checkpoint by E2 proteins from oncogenic human papillomaviruses correlates with increased turnover of the p53 tumor suppressor protein.

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Papillomavirus E2 induces senescence in HPV-positive cells via pRB- and p21(CIP)-dependent pathways

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Repression of human papillomavirus oncogenes in HeLa cervical carcinoma cells causes the orderly reactivation of dormant tumor suppressor pathways

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Repression of the integrated papillomavirus E6/E7 promoter is required for growth suppression of cervical cancer cells.

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Papillomavirus E2 induces p53-independent apoptosis in HeLa cells

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The human papillomavirus (HPV) 16 E2 protein induces apoptosis in the absence of other HPV proteins and via a p53-dependent pathway.

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Transformation and replication in mouse cells of a bovine papillomavirus--pML2 plasmid vector that can be rescued in bacteria.

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Plasmids for the in vitro analysis of RNA polymerase II-dependent transcription based on a G-free template.

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TAFII-independent activation mediated by human TBP in the presence of the positive cofactor PC4

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Properties of PC4 and an RNA polymerase II complex in directing activated and basal transcription in vitro

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Isolation of mouse TFIID and functional characterization of TBP and TFIID in mediating estrogen receptor and chromatin transcription

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Specific recognition nucleotides and their DNA context determine the affinity of E2 protein for 17 binding sites in the BPV-1 genome

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The structural basis of DNA target discrimination by papillomavirus E2 proteins

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The E2 trans-activator can act as a repressor by interfering with a cellular transcription factor

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The hinge of the human papillomavirus type 11 E2 protein contains major determinants for nuclear localization and nuclear matrix association

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Stability of the human papillomavirus type 18 E2 protein is regulated by a proteasome degradation pathway through its amino-terminal transactivation domain

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Structure of the intact transactivation domain of the human papillomavirus E2 protein

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Distinctive Cognate Sequence Discrimination, Bound DNA Conformation, and Binding Modes in the E2 C-Terminal Domains from Prototype Human and Bovine Papillomaviruses†

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Interaction of human papillomavirus 8 regulatory proteins E2, E6 and E7 with components of the TFIID complex

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Magnesium ions enhance the transfer of human papillomavirus E2 protein from non-specific to specific binding sites

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DNA structure and flexibility in the sequence-specific binding of papillomavirus E2 proteins

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Identifiers

DOI

10.1074/JBC.M206829200

1 reference

31 July 2017

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