Platelet-derived Toll-like receptor 4 (Tlr-4) is sufficient to promote microvascular thrombosis in endotoxemia. (Q34387761)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:22911769%20AND%20SRC:MED&resulttype=core&format=json

11 February 2020

title

Platelet-derived Toll-like receptor 4 (Tlr-4) is sufficient to promote microvascular thrombosis in endotoxemia (English)

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11 February 2020

0 references

1 reference

1

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11 February 2020

Rolando E Rumbaut

series ordinal

3

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11 February 2020

author name string

Niloufar Aghakasiri

series ordinal

2

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11 February 2020

language of work or name

English

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publication date

20 July 2012

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11 February 2020

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11 February 2020

volume

7

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11 February 2020

issue

7

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11 February 2020

page(s)

e41254

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Creative Commons CC0 License

11 February 2020

copyright license

start time

20 July 2012

1 reference

April 2022 Public Data File from Crossref

copyright status

copyrighted, dedicated to the public domain by copyright holder

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cites work

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Lipopolysaccharide stimulates platelet secretion and potentiates platelet aggregation via TLR4/MyD88 and the cGMP-dependent protein kinase pathway

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6 July 2018

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Platelet TLR4 activates neutrophil extracellular traps to ensnare bacteria in septic blood

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Platelet response to corneal abrasion is necessary for acute inflammation and efficient re-epithelialization

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Platelet Toll-like receptor expression modulates lipopolysaccharide-induced thrombocytopenia and tumor necrosis factor-alpha production in vivo

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Platelets express functional Toll-like receptor-4.

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Accumulation of tissue factor into developing thrombi in vivo is dependent upon microparticle P-selectin glycoprotein ligand 1 and platelet P-selectin

6 July 2018

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Endothelium-derived Toll-like receptor-4 is the key molecule in LPS-induced neutrophil sequestration into lungs

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https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0041254

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21 January 2018

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12 December 2020

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12 December 2020

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12 December 2020

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12 December 2020

Endotoxin enhances microvascular thrombosis in mouse cremaster venules via a TLR4-dependent, neutrophil-independent mechanism

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Expanding the global epidemiology of sepsis

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12 December 2020

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11 February 2020

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