Mechanism of vasodilation to adenosine in coronary arterioles from patients with heart disease. (Q34403710)

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9 January 2020

title

Mechanism of vasodilation to adenosine in coronary arterioles from patients with heart disease (English)

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9 January 2020

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8

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9 January 2020

author name string

Atsushi Sato

1

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9 January 2020

Ken Terata

2

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9 January 2020

Hiroto Miura

3

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9 January 2020

Kazuyoshi Toyama

4

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9 January 2020

Fausto R Loberiza

5

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9 January 2020

Ossama A Hatoum

6

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9 January 2020

Takashi Saito

7

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9 January 2020

David D Gutterman

9

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publication date

1 April 2005

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American Journal of Physiology Heart and Circulatory Physiology

9 January 2020

published in

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9 January 2020

volume

288

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issue

4

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page(s)

H1633-40

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Blockade of the ATP-sensitive potassium channel modulates reactive hyperemia in the canine coronary circulation

9 January 2020

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Functional and molecular characterization of receptor subtypes mediating coronary microvascular dilation to adenosine

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A human intermediate conductance calcium-activated potassium channel

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Adenosine mediates relaxation of human small resistance-like coronary arteries via A2B receptors

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Blockade of the intermediate-conductance calcium-activated potassium channel as a new therapeutic strategy for restenosis

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High glucose impairs voltage-gated K(+) channel current in rat small coronary arteries

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Adenosine as an adjunct to thrombolytic therapy for acute myocardial infarction: results of a multicenter, randomized, placebo-controlled trial: the Acute Myocardial Infarction STudy of ADenosine (AMISTAD) trial

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Pharmacologic activation of the human coronary microcirculation in vitro: endothelium-dependent dilation and differential responses to acetylcholine.

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Role for hydrogen peroxide in flow-induced dilation of human coronary arterioles

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Diabetes mellitus impairs vasodilation to hypoxia in human coronary arterioles: reduced activity of ATP-sensitive potassium channels

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

Identifiers

DOI

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9 January 2020

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