Neuronal gap junctions are required for NMDA receptor-mediated excitotoxicity: implications in ischemic stroke (Q34430454)

2 August 2017

Neuronal gap junctions are required for NMDA receptor-mediated excitotoxicity: implications in ischemic stroke (English)

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2 August 2017

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Yongfu Wang

1

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2 August 2017

Janna V Denisova

2

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2 August 2017

Ki Sung Kang

3

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2 August 2017

Joseph D Fontes

4

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2 August 2017

Bao Ting Zhu

5

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2 August 2017

Andrei B Belousov

6

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2 August 2017

language of work or name

English

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publication date

13 October 2010

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Journal of Neurophysiology

2 August 2017

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2 August 2017

104

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2 August 2017

6

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2 August 2017

3551-3556

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describes a project that uses

2 August 2017

ImageJ

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11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC3007655/fullTextXML

inferred from PubMed Central ID database lookup

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Expression and functions of neuronal gap junctions

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Potent block of Cx36 and Cx50 gap junction channels by mefloquine

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Blockade of gap junctions in vivo provides neuroprotection after perinatal global ischemia

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Impaired electrical signaling disrupts gamma frequency oscillations in connexin 36-deficient mice

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Excitatory effects of gap junction blockers on cerebral cortex seizure-like activity in rats and mice.

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Specific gap junctions enhance the neuronal vulnerability to brain traumatic injury.

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Nerve injury induces gap junctional coupling among axotomized adult motor neurons

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PubMed

https://pubmed.ncbi.nlm.nih.gov/20943940

12 December 2020

inferred from PubMed ID database lookup

Gap junctions are required for NMDA receptor dependent cell death in developing neurons

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/20943940

12 December 2020

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Occurrence of D-aspartic acid and N-methyl-D-aspartic acid in rat neuroendocrine tissues and their role in the modulation of luteinizing hormone and growth hormone release

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PubMed

https://pubmed.ncbi.nlm.nih.gov/20943940

12 December 2020

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Identifiers

DOI

10.1152/JN.00656.2010

1 reference

2 August 2017

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2 August 2017

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