How glucocorticoids control their own strength and the balance between pro- and anti-inflammatory mediators (Q34473404)

2 August 2017

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title

How glucocorticoids control their own strength and the balance between pro- and anti-inflammatory mediators (English)

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2 August 2017

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2

Claude Libert

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2 August 2017

Wim Van Molle

1

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PubMed ID

16331703

2 August 2017

language of work or name

English

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publication date

1 December 2005

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European Journal of Immunology

2 August 2017

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2 August 2017

35

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2 August 2017

12

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2 August 2017

3396-3399

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Molecular mechanisms of glucocorticoid action: what is important?

2 August 2017

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Drug-induced bone loss

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Renal transplant recipient attitudes toward steroid use and steroid withdrawal

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Corticosteroid-insensitive asthma: molecular mechanisms.

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The use of corticosteroids in severe sepsis and acute respiratory distress syndrome

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Mechanism of a Reaction in Vitro Associated with Delayed-Type Hypersensitivity

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MIF is a pituitary-derived cytokine that potentiates lethal endotoxaemia

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Targeted disruption of migration inhibitory factor gene reveals its critical role in sepsis.

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Macrophage migration inhibitory factor is a critical mediator of the activation of immune cells by exotoxins of Gram-positive bacteria

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Migration-inhibitory factor gene-deficient mice are susceptible to cutaneous Leishmania major infection

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MIF as a glucocorticoid-induced modulator of cytokine production

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Macrophage migration inhibitory factor promotes innate immune responses by suppressing glucocorticoid-induced expression of mitogen-activated protein kinase phosphatase-1.

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New insights into the control of MAP kinase pathways

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MK2 targets AU-rich elements and regulates biosynthesis of tumor necrosis factor and interleukin-6 independently at different post-transcriptional levels

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MAP kinase phosphatase 1: a novel mediator of biological effects of glucocorticoids?

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7 January 2021

Nuclear export of glucocorticoid receptor is enhanced by c-Jun N-terminal kinase-mediated phosphorylation

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https://api.crossref.org/works/10.1002%2FEJI.200535556

7 January 2021

p38 Mitogen-activated protein kinase-induced glucocorticoid receptor phosphorylation reduces its activity: role in steroid-insensitive asthma

1 reference

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Superantigen-induced corticosteroid resistance of human T cells occurs through activation of the mitogen-activated protein kinase kinase/extracellular signal-regulated kinase (MEK-ERK) pathway

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Preheating accelerates mitogen-activated protein (MAP) kinase inactivation post-heat shock via a heat shock protein 70-mediated increase in phosphorylated MAP kinase phosphatase-1.

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7 January 2021

Identifiers

DOI

10.1002/EJI.200535556

1 reference

2 August 2017

1 reference

PubMed ID

16331703