Sustained phosphorylation of Bid is a marker for resistance to Fas-induced apoptosis during chronic liver diseases. (Q34504973)

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15 January 2020

title

Sustained phosphorylation of Bid is a marker for resistance to Fas-induced apoptosis during chronic liver diseases (English)

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15 January 2020

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chronic liver disease

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author

Joseph E. Aslan

2

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15 January 2020

Markus Grompe

8

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15 January 2020

author name string

Arndt Vogel

1

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15 January 2020

Holger Willenbring

3

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15 January 2020

Christian Klein

4

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15 January 2020

Milton Finegold

5

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15 January 2020

Howard Mount

6

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15 January 2020

Gary Thomas

7

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15 January 2020

language of work or name

English

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publication date

1 January 2006

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15 January 2020

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15 January 2020

volume

130

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15 January 2020

issue

1

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15 January 2020

page(s)

104-119

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PACS-2 controls endoplasmic reticulum-mitochondria communication and Bid-mediated apoptosis

15 January 2020

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Post-translational modification of Bid has differential effects on its susceptibility to cleavage by caspase 8 or caspase 3.

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The expanding role of mitochondria in apoptosis

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Phosphorylation of bid by casein kinases I and II regulates its cleavage by caspase 8.

7 July 2018

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7 July 2018

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Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

7 July 2018

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Cellular FLICE-inhibitory protein splice variants inhibit different steps of caspase-8 activation at the CD95 death-inducing signaling complex

7 July 2018

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Bile acid-induced rat hepatocyte apoptosis is inhibited by antioxidants and blockers of the mitochondrial permeability transition

7 July 2018

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Posttranslational N-myristoylation of BID as a molecular switch for targeting mitochondria and apoptosis

7 July 2018

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tBID, a membrane-targeted death ligand, oligomerizes BAK to release cytochrome c

7 July 2018

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Fas preassociation required for apoptosis signaling and dominant inhibition by pathogenic mutations

7 July 2018

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Damage-mediated phosphorylation of human p53 threonine 18 through a cascade mediated by a casein 1-like kinase. Effect on Mdm2 binding

7 July 2018

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NFkappaB mediates apoptosis through transcriptional activation of Fas (CD95) in adenoviral hepatitis

7 July 2018

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Bid induces the oligomerization and insertion of Bax into the outer mitochondrial membrane

7 July 2018

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7 July 2018

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Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

7 July 2018

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Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

7 July 2018

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7 July 2018

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Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death

7 July 2018

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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

7 July 2018

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Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

7 July 2018

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Hepatocyte injury in tyrosinemia type 1 is induced by fumarylacetoacetate and is inhibited by caspase inhibitors

7 July 2018

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7 July 2018

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7 July 2018

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p53 is phosphorylated in vitro and in vivo by the delta and epsilon isoforms of casein kinase 1 and enhances the level of casein kinase 1 delta in response to topoisomerase-directed drugs

7 July 2018

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Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis

7 July 2018

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Suppression of TNF-alpha-induced apoptosis by NF-kappaB

7 July 2018

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7 July 2018

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Loss of fumarylacetoacetate hydrolase is responsible for the neonatal hepatic dysfunction phenotype of lethal albino mice.

7 July 2018

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7 July 2018

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Casein kinase-1 phosphorylates the p75 tumor necrosis factor receptor and negatively regulates tumor necrosis factor signaling for apoptosis

7 July 2018

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7 July 2018

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7 July 2018

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7 July 2018

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27 October 2018

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27 October 2018

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Chronic liver disease in murine hereditary tyrosinemia type 1 induces resistance to cell death.

27 October 2018

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Resistance of rat hepatocytes against bile acid-induced apoptosis in cholestatic liver injury is due to nuclear factor-kappa B activation

27 October 2018

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27 October 2018

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Activation of caspases and cleavage of Bid are required for tyrosine and phenylalanine deficiency-induced apoptosis of human A375 melanoma cells

27 October 2018

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27 October 2018

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Bid antisense attenuates bile acid-induced apoptosis and cholestatic liver injury.

27 October 2018

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27 October 2018

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27 October 2018

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27 October 2018

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Tumor necrosis factor alpha prevents tumor necrosis factor receptor-mediated mouse hepatocyte apoptosis, but not fas-mediated apoptosis: role of nuclear factor-kappaB

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Bcl-2 family member Bfl-1/A1 sequesters truncated bid to inhibit is collaboration with pro-apoptotic Bak or Bax

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