Programmed cell death 1 (PD-1) and its ligand PD-L1 are required for allograft tolerance (Q34583370)

3 August 2017

title

Programmed cell death 1 (PD-1) and its ligand PD-L1 are required for allograft tolerance (English)

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3 August 2017

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Liqing Wang

1

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3 August 2017

Rongxiang Han

series ordinal

2

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3 August 2017

Wayne W Hancock

series ordinal

3

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3 August 2017

language of work or name

English

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publication date

1 October 2007

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European Journal of Immunology

3 August 2017

published in

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3 August 2017

volume

37

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3 August 2017

issue

10

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3 August 2017

page(s)

2983-2990

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Programmed death-1 targeting can promote allograft survival

3 August 2017

cites work

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Differential effects of B and T lymphocyte attenuator and programmed death-1 on acceptance of partially versus fully MHC-mismatched cardiac allografts.

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Blocking the monocyte chemoattractant protein-1/CCR2 chemokine pathway induces permanent survival of islet allografts through a programmed death-1 ligand-1-dependent mechanism

21 January 2018

1 reference

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Tumor-associated B7-H1 promotes T-cell apoptosis: a potential mechanism of immune evasion

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Regulated compartmentalization of programmed cell death-1 discriminates CD4+CD25+ resting regulatory T cells from activated T cells.

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7 January 2021

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7 January 2021

B7-H1, a third member of the B7 family, co-stimulates T-cell proliferation and interleukin-10 secretion

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7 January 2021

PD-L2 is a second ligand for PD-1 and inhibits T cell activation

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7 January 2021

CTLA-4 and PD-1 receptors inhibit T-cell activation by distinct mechanisms

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7 January 2021

Development of lupus-like autoimmune diseases by disruption of the PD-1 gene encoding an ITIM motif-carrying immunoreceptor

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Establishment of NOD-Pdcd1-/- mice as an efficient animal model of type I diabetes

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The programmed death-1 (PD-1) pathway regulates autoimmune diabetes in nonobese diabetic (NOD) mice

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Differential role of programmed death-ligand 1 [corrected] and programmed death-ligand 2 [corrected] in regulating the susceptibility and chronic progression of experimental autoimmune encephalomyelitis

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7 January 2021

B7-H1 costimulation preferentially enhances CD28-independent T-helper cell function

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7 January 2021

Chemokine receptor‐dependent alloresponses

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7 January 2021

Requirement of the chemokine receptor CXCR3 for acute allograft rejection

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7 January 2021

Donor-derived IP-10 initiates development of acute allograft rejection

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7 January 2021

Beneficial effects of targeting CCR5 in allograft recipients

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IDO expression by dendritic cells: tolerance and tryptophan catabolism

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7 January 2021

Antibody-induced transplant arteriosclerosis is prevented by graft expression of anti-oxidant and anti-apoptotic genes

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7 January 2021

Restoring function in exhausted CD8 T cells during chronic viral infection

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7 January 2021

Upregulation of PD-1 Expression on Circulating and Intrahepatic Hepatitis C Virus-Specific CD8+ T Cells Associated with Reversible Immune Dysfunction

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7 January 2021

Contribution of the PD-L1/PD-1 pathway to T-cell exhaustion: an update on implications for chronic infections and tumor evasion

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7 January 2021

Involvement of the programmed death-1/programmed death-1 ligand pathway in CD4+CD25+ regulatory T-cell activity to suppress alloimmune responses

1 reference

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7 January 2021

Treg suppressive activity involves estrogen-dependent expression of programmed death-1 (PD-1)

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7 January 2021

CD4+CD25high regulatory cells in human peripheral blood

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7 January 2021

Genetics of autoimmune diseases--disorders of immune homeostasis

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7 January 2021

Importance of ICOS-B7RP-1 costimulation in acute and chronic allograft rejection

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7 January 2021

Identifiers

DOI

10.1002/EJI.200737583

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3 August 2017

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