Reactive oxygen species cause endothelial dysfunction in chronic flow overload (Q34598465)

3 August 2017

title

Reactive oxygen species cause endothelial dysfunction in chronic flow overload (English)

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3 August 2017

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reactive oxygen species

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X Lu

1

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3 August 2017

X Guo

2

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3 August 2017

C D Wassall

3

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3 August 2017

M D Kemple

4

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3 August 2017

J L Unthank

5

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3 August 2017

G S Kassab

6

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3 August 2017

language of work or name

English

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publication date

2 December 2010

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Journal of Applied Physiology

3 August 2017

published in

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3 August 2017

volume

110

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3 August 2017

issue

2

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3 August 2017

page(s)

520-527

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Apocynin is not an inhibitor of vascular NADPH oxidases but an antioxidant.

3 August 2017

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An endothelium-derived hyperpolarizing factor distinct from NO and prostacyclin is a major endothelium-dependent vasodilator in resistance vessels of wild-type and endothelial NO synthase knockout mice.

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An NADPH oxidase superoxide-generating system in the rabbit aorta

7 July 2018

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21 January 2018

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Stretch enhances contraction of bovine coronary arteries via an NAD(P)H oxidase-mediated activation of the extracellular signal-regulated kinase mitogen-activated protein kinase cascade

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12 December 2020

Shear level influences resistance artery remodeling: wall dimensions, cell density, and eNOS expression

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12 December 2020

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12 December 2020

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12 December 2020

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12 December 2020

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12 December 2020

NADPH Oxidase Accounts for Enhanced Superoxide Production and Impaired Endothelium-Dependent Smooth Muscle Relaxation in BKβ1 −/− Mice

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12 December 2020

Apocynin normalizes hyperreactivity to phenylephrine in mesenteric arteries from cholesterol-fed mice by improving endothelium-derived hyperpolarizing factor response

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12 December 2020

Flow-induced remodeling in resistance arteries from obese Zucker rats is associated with endothelial dysfunction

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12 December 2020

10.1152/JAPPLPHYSIOL.00786.2009

Identifiers

DOI

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3 August 2017

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3 August 2017

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