DNA variation in the SNAP25 gene confers risk to ADHD and is associated with reduced expression in prefrontal cortex (Q34672850)

3 August 2017

title

DNA variation in the SNAP25 gene confers risk to ADHD and is associated with reduced expression in prefrontal cortex (English)

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3 August 2017

8

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3 August 2017

9

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3 August 2017

Ziarih Hawi

1

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Lindsey Kent

object named as

Lindsey Kent

7

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Alasdair Vance

object named as

Alasdair Vance

6

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author name string

Natasha Matthews

2

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3 August 2017

Joseph Wagner

3

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3 August 2017

Robyn H Wallace

4

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3 August 2017

Tim J Butler

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3 August 2017

language of work or name

English

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publication date

12 April 2013

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3 August 2017

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3 August 2017

volume

8

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3 August 2017

issue

4

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3 August 2017

page(s)

e60274

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Creative Commons Attribution 4.0 International

3 August 2017

copyright license

start time

12 April 2013

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April 2022 Public Data File from Crossref

copyright status

copyrighted

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cites work

Catecholamine influences on prefrontal cortical function: relevance to treatment of attention deficit/hyperactivity disorder and related disorders

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Family-based genome-wide association scan of attention-deficit/hyperactivity disorder

7 July 2018

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Helical extension of the neuronal SNARE complex into the membrane

7 July 2018

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Candidate gene studies of ADHD: a meta-analytic review

7 July 2018

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Genome-wide association scan of the time to onset of attention deficit hyperactivity disorder

7 July 2018

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Genome-wide association scan of quantitative traits for attention deficit hyperactivity disorder identifies novel associations and confirms candidate gene associations

7 July 2018

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The analysis of 51 genes in DSM-IV combined type attention deficit hyperactivity disorder: association signals in DRD4, DAT1 and 16 other genes

7 July 2018

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Activation in ventral prefrontal cortex is sensitive to genetic vulnerability for attention-deficit hyperactivity disorder

7 July 2018

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Synaptosomal-associated protein 25 (SNAP-25) and attention deficit hyperactivity disorder (ADHD): evidence of linkage and association in the Irish population

7 July 2018

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The structure of haplotype blocks in the human genome

7 July 2018

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Association study of a SNAP-25 microsatellite and attention deficit hyperactivity disorder

7 July 2018

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Embryonic lethality and vascular defects in mice lacking the Notch ligand Jagged1

7 July 2018

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Radiation hybrid mapping of SNAP, PCSK2, and THBD (human chromosome 20p)

7 July 2018

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SNAP receptors implicated in vesicle targeting and fusion

7 July 2018

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The identification of a novel synaptosomal-associated protein, SNAP-25, differentially expressed by neuronal subpopulations

7 July 2018

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Expression of catecholaminergic mRNAs in the hyperactive mouse mutant coloboma

7 July 2018

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Norepinephrine regulates locomotor hyperactivity in the mouse mutant coloboma

21 January 2018

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Haplotype analysis of SNAP-25 suggests a role in the aetiology of ADHD.

21 January 2018

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The SNAP25 gene as a susceptibility gene contributing to attention-deficit hyperactivity disorder.

21 January 2018

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Regional meta-analysis of published data supports linkage of autism with markers on chromosome 7.

21 January 2018

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Role of SNAP25 explored in eastern Indian attention deficit hyperactivity disorder probands.

21 January 2018

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https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0060274

Allelic variants of SNAP25 in a family-based sample of ADHD.

21 January 2018

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https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0060274

Candidate gene analysis in an on-going genome-wide association study of attention-deficit hyperactivity disorder: suggestive association signals in ADRA1A.

21 January 2018

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Cortical abnormalities in children and adolescents with attention-deficit hyperactivity disorder

21 January 2018

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Phenotype studies of the DRD4 gene polymorphisms in ADHD: association with oppositional defiant disorder and positive family history

21 January 2018

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Abnormal spatial asymmetry of selective attention in ADHD.

21 January 2018

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Evidence that variation at the serotonin transporter gene influences susceptibility to attention deficit hyperactivity disorder (ADHD): analysis and pooled analysis

21 January 2018

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https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0060274

Inhibition of axonal growth by SNAP-25 antisense oligonucleotides in vitro and in vivo

21 January 2018

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0060274

Haplotyping of putative microRNA-binding sites in the SNAP-25 gene.

21 January 2018

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0060274

Sequential N- to C-terminal SNARE complex assembly drives priming and fusion of secretory vesicles

21 January 2018

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0060274

A high-density single-nucleotide polymorphism screen of 23 candidate genes in attention deficit hyperactivity disorder: suggesting multiple susceptibility genes among Chinese Han population

21 January 2018

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0060274

Investigation of variation inSNAP-25 and ADHD and relationship to co-morbid major depressive disorder

21 January 2018

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0060274

Biased paternal transmission of SNAP-25 risk alleles in attention-deficit hyperactivity disorder

21 January 2018

1 reference

12 December 2020

Deletion map of the coloboma (Cm) locus on mouse chromosome 2

1 reference

12 December 2020

Identification of DNA variants in the SNAP-25 gene and linkage study of these polymorphisms and attention-deficit hyperactivity disorder

1 reference

12 December 2020

Mouse model of hyperkinesis implicates SNAP-25 in behavioral regulation

1 reference

12 December 2020

Support for the MnlI polymorphism of SNAP25; a Korean ADHD case-control study

1 reference

12 December 2020

10.1371/JOURNAL.PONE.0060274

Identifiers

DOI

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3 August 2017

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3 August 2017

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