Class A scavenger receptor 1 (MSR1) restricts hepatitis C virus replication by mediating toll-like receptor 3 recognition of viral RNAs produced in neighboring cells. (Q34743740)

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English	Class A scavenger receptor 1 (MSR1) restricts hepatitis C virus replication by mediating toll-like receptor 3 recognition of viral RNAs produced in neighboring cells.	scientific article

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2 March 2020

Class A scavenger receptor 1 (MSR1) restricts hepatitis C virus replication by mediating toll-like receptor 3 recognition of viral RNAs produced in neighboring cells (English)

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2 March 2020

toll-like receptor

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Hepatitis C virus

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based on heuristic

inferred from title

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based on heuristic

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David R. McGivern

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author name string

Hiromichi Dansako

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2 March 2020

Daisuke Yamane

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Christoph Welsch

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Fengyu Hu

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Nobuyuki Kato

6

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Stanley M Lemon

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publication date

23 May 2013

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9

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5

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e1003345

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copyright license

Creative Commons Attribution 4.0 International

23 May 2013

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April 2022 Public Data File from Crossref

copyright status

0 references

Competing and noncompeting activities of miR-122 and the 5' exonuclease Xrn1 in regulation of hepatitis C virus replication.

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Base pairing between hepatitis C virus RNA and microRNA 122 3' of its seed sequence is essential for genome stabilization and production of infectious virus.

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Activation of chemokine and inflammatory cytokine response in hepatitis C virus-infected hepatocytes depends on Toll-like receptor 3 sensing of hepatitis C virus double-stranded RNA intermediates

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Hepatitis C virus infection causes cell cycle arrest at the level of initiation of mitosis

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Hepatitis C virus NS2 protein serves as a scaffold for virus assembly by interacting with both structural and nonstructural proteins

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Scavenger receptor A1 is required for sensing HCMV by endosomal TLR-3/-9 in monocytic THP-1 cells

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Toll-Like Receptor 3 Mediates Establishment of an Antiviral State against Hepatitis C Virus in Hepatoma Cells

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Double-stranded RNA-induced interferon-beta and inflammatory cytokine production modulated by hepatitis C virus serine proteases derived from patients with hepatic diseases

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Toll-Like Receptors and Viruses: Induction of Innate Antiviral Immune Responses

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7 July 2018

Innate immunity induced by composition-dependent RIG-I recognition of hepatitis C virus RNA

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7 July 2018

NS3 Helicase Domains Involved in Infectious Intracellular Hepatitis C Virus Particle Assembly

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Interferon signaling and treatment outcome in chronic hepatitis C

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Visualization of Double-Stranded RNA in Cells Supporting Hepatitis C Virus RNA Replication

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Scavenger receptor class-A is a novel cell surface receptor for double-stranded RNA.

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Limited suppression of the interferon-beta production by hepatitis C virus serine protease in cultured human hepatocytes

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Compensatory Mutations in E1, p7, NS2, and NS3 Enhance Yields of Cell Culture-Infectious Intergenotypic Chimeric Hepatitis C Virus

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5'-Triphosphate RNA is the ligand for RIG-I

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Toll-like receptor 3 associates with c-Src tyrosine kinase on endosomes to initiate antiviral signaling

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Hepatitis C virus protease NS3/4A cleaves mitochondrial antiviral signaling protein off the mitochondria to evade innate immunity

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Cardif is an adaptor protein in the RIG-I antiviral pathway and is targeted by hepatitis C virus

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Recognition of double-stranded RNA by human toll-like receptor 3 and downstream receptor signaling requires multimerization and an acidic pH.

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Charged collagen structure mediates the recognition of negatively charged macromolecules by macrophage scavenger receptors.

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A role for macrophage scavenger receptors in atherosclerosis and susceptibility to infection

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The histidine interruption of an alpha-helical coiled coil allosterically mediates a pH-dependent ligand dissociation from macrophage scavenger receptors

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based on heuristic

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Acid destabilization of a triple-helical peptide model of the macrophage scavenger receptor

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https://pubmed.ncbi.nlm.nih.gov/23717201

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based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1371/JOURNAL.PPAT.1003345

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2 March 2020

1 reference

Europe PubMed Central

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2 March 2020

1 reference

Europe PubMed Central

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2 March 2020

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