Mice with hypomorphic expression of the sodium-phosphate cotransporter PiT1/Slc20a1 have an unexpected normal bone mineralization. (Q34778791)

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3 March 2020

title

Mice with hypomorphic expression of the sodium-phosphate cotransporter PiT1/Slc20a1 have an unexpected normal bone mineralization (English)

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3 March 2020

9

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3 March 2020

Jérome Guicheux

10

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3 March 2020

Laurent Beck

11

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3 March 2020

author name string

Annabelle Bourgine

1

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3 March 2020

Paul Pilet

2

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Sara Diouani

3

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Sophie Sourice

4

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Julie Lesoeur

5

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Sarah Beck-Cormier

6

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Solmaz Khoshniat

7

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Pierre Weiss

8

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language of work or name

English

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publication date

13 June 2013

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3 March 2020

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3 March 2020

volume

8

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issue

6

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3 March 2020

page(s)

e65979

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Creative Commons Attribution 4.0 International

3 March 2020

copyright license

start time

13 June 2013

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April 2022 Public Data File from Crossref

copyright status

copyrighted

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cites work

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Characterization of transport mechanisms and determinants critical for Na+-dependent Pi symport of the PiT family paralogs human PiT1 and PiT2

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Enhanced expression of the inorganic phosphate transporter Pit-1 is involved in BMP-2-induced matrix mineralization in osteoblast-like cells.

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Stimulation of sodium-dependent inorganic phosphate transport by activation of Gi/o-protein-coupled receptors by epinephrine in MC3T3-E1 osteoblast-like cells

7 July 2018

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Aggregation of hydroxyapatite crystals

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12 December 2020

Expression of a newly identified phosphate transporter/retrovirus receptor in human SaOS-2 osteoblast-like cells and its regulation by insulin-like growth factor I

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The mechanism of matrix vesicle formation. Studies on the composition of chondrocyte microvilli and on the effects of microfilament-perturbing agents on cellular vesiculation

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In vivo expression of transcripts encoding the Glvr-1 phosphate transporter/retrovirus receptor during bone development

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Pro-Inflammatory Cytokines Produced by Growth Plate Chondrocytes May Act Locally to Modulate Longitudinal Bone Growth

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10.1371/JOURNAL.PONE.0065979

Identifiers

DOI

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3 March 2020

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3 March 2020

PubMed publication ID

23785462

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