Angiotensin II-induced vascular endothelial dysfunction through RhoA/Rho kinase/p38 mitogen-activated protein kinase/arginase pathway (Q34979275)

5 August 2017

title

Angiotensin II-induced vascular endothelial dysfunction through RhoA/Rho kinase/p38 mitogen-activated protein kinase/arginase pathway (English)

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5 August 2017

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Alia Shatanawi

1

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5 August 2017

Maritza J Romero

2

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5 August 2017

Jennifer A Iddings

3

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5 August 2017

Surabhi Chandra

4

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5 August 2017

Nagavedi S Umapathy

5

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5 August 2017

Alexander D Verin

6

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5 August 2017

Ruth B Caldwell

7

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5 August 2017

R William Caldwell

8

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5 August 2017

publication date

2 February 2011

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American Journal of Physiology - Cell Physiology

5 August 2017

published in

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5 August 2017

volume

300

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5 August 2017

issue

5

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5 August 2017

page(s)

C1181-92

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describes a project that uses

5 August 2017

ImageJ

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11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC3093945/fullTextXML

inferred from PubMed Central ID database lookup

cites work

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12 December 2020

Pharmacological profile of SB 203580, a selective inhibitor of cytokine suppressive binding protein/p38 kinase, in animal models of arthritis, bone resorption, endotoxin shock and immune function

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A betagamma dimer derived from G13 transduces the angiotensin AT1 receptor signal to stimulation of Ca2+ channels in rat portal vein myocytes

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Increased arginase activity and endothelial dysfunction in human inflammatory bowel disease

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Effect of the angiotensin II type 1 receptor blocker candesartan on endothelial function in patients with essential hypertension

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SB 239063, a potent p38 MAP kinase inhibitor, reduces inflammatory cytokine production, airways eosinophil infiltration, and persistence

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Identifiers

DOI

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5 August 2017

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5 August 2017

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