Type I IFN signaling triggers immunopathology in tuberculosis-susceptible mice by modulating lung phagocyte dynamics (Q34984025)

5 August 2017

Type I IFN signaling triggers immunopathology in tuberculosis-susceptible mice by modulating lung phagocyte dynamics (English)

1 reference

5 August 2017

0 references

Hans-Joachim Mollenkopf

12

Hans-Joachim Mollenkopf

0 references

Vladimir Yeremeev

Vladimir Yeremeev

2

0 references

Stefan Kaufmann

Stefan H E Kaufmann

14

0 references

Alexis Vogelzang

Alexis Vogelzang

9

0 references

January Weiner

4

January Weiner

1 reference

5 August 2017

Anca Dorhoi

1

1 reference

5 August 2017

Geraldine Nouailles

3

1 reference

5 August 2017

Sabine Jörg

5

1 reference

5 August 2017

Ellen Heinemann

6

1 reference

5 August 2017

Dagmar Oberbeck-Müller

7

1 reference

5 August 2017

Julia K Knaul

8

1 reference

5 August 2017

Stephen T Reece

10

1 reference

5 August 2017

Karin Hahnke

11

1 reference

5 August 2017

Volker Brinkmann

13

1 reference

5 August 2017

language of work or name

English

0 references

publication date

24 June 2014

1 reference

European Journal of Immunology

5 August 2017

1 reference

5 August 2017

44

1 reference

5 August 2017

2380-2393

1 reference

5 August 2017

8

1 reference

describes a project that uses

5 August 2017

limma

1 reference

22 June 2022

https://europepmc.org/article/PMC/4298793

based on heuristic

inferred from PubMed Central ID database lookup

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Intranasal Poly-IC treatment exacerbates tuberculosis in mice through the pulmonary recruitment of a pathogen-permissive monocyte/macrophage population

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The adaptor molecule CARD9 is essential for tuberculosis control

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Type I interferon modulates monocyte recruitment and maturation in chronic inflammation

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Type I IFNs mediate development of postinfluenza bacterial pneumonia in mice

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A novel signaling pathway impact analysis.

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Type I IFNs enhance susceptibility to Chlamydia muridarum lung infection by enhancing apoptosis of local macrophages

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Virulence of a Mycobacterium tuberculosis clinical isolate in mice is determined by failure to induce Th1 type immunity and is associated with induction of IFN-alpha /beta

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PubMed

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12 December 2020

based on heuristic

[Interferon alpha-2b in the complex therapy of patients with pulmonary tuberculosis concurrent with bronchoobstructive syndrome]

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/24782112

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1002/EJI.201344219

1 reference

5 August 2017

1 reference

5 August 2017

1 reference