Menin interacting proteins as clues toward the understanding of multiple endocrine neoplasia type 1. (Q35006210)

5 August 2017

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title

Menin interacting proteins as clues toward the understanding of multiple endocrine neoplasia type 1. (English)

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multiple endocrine neoplasia

5 August 2017

main subject

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Ariane Poisson

1

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5 August 2017

Barbara Zablewska

series ordinal

2

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5 August 2017

Patrick Gaudray

series ordinal

3

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5 August 2017

language of work or name

English

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publication date

1 January 2003

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5 August 2017

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5 August 2017

volume

189

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5 August 2017

issue

1

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5 August 2017

page(s)

1-10

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Positional cloning of the gene for multiple endocrine neoplasia-type 1

5 August 2017

cites work

1 reference

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7 January 2021

Identification of the multiple endocrine neoplasia type 1 (MEN1) gene. The European Consortium on MEN1

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7 January 2021

Characterization of the mouse Men1 gene and its expression during development

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7 January 2021

Characterization of a MEN1 ortholog from Drosophila melanogaster

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7 January 2021

Characterization of the MEN1 ortholog in zebrafish

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7 January 2021

Germline mutation profile of MEN1 in multiple endocrine neoplasia type 1: search for correlation between phenotype and the functional domains of the MEN1 protein.

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7 January 2021

Expression analysis of endogenous menin, the product of the multiple endocrine neoplasia type 1 gene, in cell lines and human tissues

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7 January 2021

Multiple endocrine neoplasia type 1 gene maps to chromosome 11 and is lost in insulinoma.

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Unstable chromosomes in heritable tumor syndromes. Multiple endocrine neoplasia type 1 (MEN1)

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7 January 2021

A mouse model of multiple endocrine neoplasia, type 1, develops multiple endocrine tumors

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7 January 2021

Menin interacts with the AP1 transcription factor JunD and represses JunD-activated transcription

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https://api.crossref.org/works/10.1016%2FS0304-3835%2802%2900509-8

7 January 2021

Menin represses JunD-activated transcription by a histone deacetylase-dependent mechanism

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7 January 2021

Stable overexpression of MEN1 suppresses tumorigenicity of RAS.

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7 January 2021

JunD protects cells from p53-dependent senescence and apoptosis.

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7 January 2021

The tumor suppressor protein menin interacts with NF-kappaB proteins and inhibits NF-kappaB-mediated transactivation

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7 January 2021

Rel/NF-kappa B/I kappa B signal transduction in the generation and treatment of human cancer

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7 January 2021

Functional cooperation between JunD and NF-kappaB in rat hepatocytes.

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7 January 2021

Inactivation of menin, a Smad3-interacting protein, blocks transforming growth factor type beta signaling

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7 January 2021

Role of Ras and Mapks in TGFbeta signaling

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7 January 2021

Menin interacts directly with the homeobox-containing protein Pem

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7 January 2021

Menin, the product of the MEN1 gene, is a nuclear protein

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3835%2802%2900509-8

7 January 2021

Menin's interaction with glial fibrillary acidic protein and vimentin suggests a role for the intermediate filament network in regulating menin activity

1 reference

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7 January 2021

Cytoplasmic localization of wild-type p53 in glioblastomas correlates with expression of vimentin and glial fibrillary acidic protein

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Evidence for a novel gene associated with low tumor metastatic potential

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7 January 2021

Nucleoside diphosphate kinase beta (Nm23-R1/NDPKbeta) is associated with intermediate filaments and becomes upregulated upon cAMP-induced differentiation of rat C6 glioma

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7 January 2021

Menin, a gene product responsible for multiple endocrine neoplasia type 1, interacts with the putative tumor metastasis suppressor nm23

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3835%2802%2900509-8

7 January 2021

Menin, the multiple endocrine neoplasia type 1 gene product, exhibits GTP-hydrolyzing activity in the presence of the tumor metastasis suppressor nm23

1 reference

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7 January 2021

Synapse formation between central neurons requires postsynaptic expression of the MEN1 tumor suppressor gene.

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7 January 2021

Identification of the tumor metastasis suppressor Nm23-H1/Nm23-R1 as a constituent of the centrosome

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7 January 2021

Chromosomal breakage in multiple endocrine adenomatosis (types I and II)

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7 January 2021

Chromosome abnormalities in lymphocytes and fibroblasts of subjects with multiple endocrine neoplasia type 1

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7 January 2021

Cytogenetics of multiple endocrine neoplasia syndrome. II. Chromosome abnormalities in an insulinoma and a glucagonoma from two subjects with MEN1

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7 January 2021

Chromosomal instability in multiple endocrine neoplasia type 1. Cytogenetic evaluation with DEB test

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7 January 2021

Premature centromere division in patients with multiple endocrine neoplasia type 1

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7 January 2021

Enhanced sensitivity to alkylating agent in lymphocytes from patients with multiple endocrine neoplasia type 1

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7 January 2021

Targeted disruption of the p50 subunit of NF-kappa B leads to multifocal defects in immune responses

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7 January 2021

B lymphocytes differentially use the Rel and nuclear factor kappaB1 (NF-kappaB1) transcription factors to regulate cell cycle progression and apoptosis in quiescent and mitogen-activated cells

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7 January 2021

Nuclear factor (NF)-kappa B2 (p100/p52) is required for normal splenic microarchitecture and B cell-mediated immune responses

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7 January 2021

Mice deficient in nuclear factor (NF)-kappa B/p52 present with defects in humoral responses, germinal center reactions, and splenic microarchitecture

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7 January 2021

Targeted disruption of Smad3 reveals an essential role in transforming growth factor beta-mediated signal transduction

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7 January 2021

Targeted disruption of SMAD3 results in impaired mucosal immunity and diminished T cell responsiveness to TGF-beta

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7 January 2021

Mice lacking Smad3 show accelerated wound healing and an impaired local inflammatory response

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7 January 2021

Functions of mammalian Smad genes as revealed by targeted gene disruption in mice

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7 January 2021

Role of the intermediate filament protein vimentin in delaying senescence and in the spontaneous immortalization of mouse embryo fibroblasts.

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7 January 2021

Effect of elevated K(+), hypotonic stress, and cortical spreading depression on astrocyte swelling in GFAP-deficient mice.

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7 January 2021

Normal reproductive and macrophage function in Pem homeobox gene-deficient mice

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7 January 2021

10.1016/S0304-3835(02)00509-8

Identifiers

DOI

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5 August 2017

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