Reduction of tumstatin in asthmatic airways contributes to angiogenesis, inflammation, and hyperresponsiveness (Q35010424)

5 August 2017

title

Reduction of tumstatin in asthmatic airways contributes to angiogenesis, inflammation, and hyperresponsiveness (English)

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5 August 2017

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Brian G Oliver

12

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Judith Lee Black

11

object named as

Judith L Black

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5 August 2017

Janette K Burgess

1

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Sarah Boustany

2

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Lyn M Moir

3

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Markus Weckmann

4

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Justine Y Lau

5

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Karryn Grafton

6

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Melissa Baraket

7

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Philip M Hansbro

8

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Nicole G Hansbro

9

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Paul S Foster

10

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language of work or name

English

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publication date

29 October 2009

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American Journal of Respiratory and Critical Care Medicine

5 August 2017

published in

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volume

181

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issue

2

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5 August 2017

page(s)

106-115

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Vascular remodeling is a feature of asthma and nonasthmatic eosinophilic bronchitis

5 August 2017

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A comparison of airway and serum matrix metalloproteinase-9 activity among normal subjects, asthmatic patients, and patients with asthmatic mucus hypersecretion.

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Physiological levels of tumstatin, a fragment of collagen IV alpha3 chain, are generated by MMP-9 proteolysis and suppress angiogenesis via alphaV beta3 integrin

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Immunostimulatory DNA inhibits allergen-induced peribronchial angiogenesis in mice

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Tumstatin, the NC1 domain of alpha3 chain of type IV collagen, is an endogenous inhibitor of pathological angiogenesis and suppresses tumor growth

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Identifiers

DOI

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5 August 2017

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