Hypoxia-like tissue injury as a component of multiple sclerosis lesions (Q35057243)

6 August 2017

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title

Hypoxia-like tissue injury as a component of multiple sclerosis lesions (English)

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6 August 2017

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Hans Lassmann

1

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6 August 2017

publication date

1 February 2003

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Journal of the Neurological Sciences

6 August 2017

published in

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6 August 2017

volume

206

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6 August 2017

issue

2

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6 August 2017

page(s)

187-191

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A quantitative analysis of oligodendrocytes in multiple sclerosis lesions. A study of 113 cases

6 August 2017

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7 January 2021

inferred from DOI database lookup

Hypoxia response element of the human vascular endothelial growth factor gene mediates transcriptional regulation by nitric oxide: control of hypoxia-inducible factor-1 activity by nitric oxide

1 reference

12 December 2020

Immunocytochemical characterization of the expression of inducible and constitutive isoforms of nitric oxide synthase in demyelinating multiple sclerosis lesions

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12 December 2020

Microglial cell cytotoxicity of oligodendrocytes is mediated through nitric oxide

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12 December 2020

Apoliprotein E and multiple sclerosis: impact of the epsilon-4 allele on susceptibility, clinical type and progression rate

1 reference

12 December 2020

Intrathecal application of interferon gamma. Progressive appearance of MHC antigens within the rat nervous system

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12 December 2020

Lesion development in Marburg's type of acute multiple sclerosis: from inflammation to demyelination

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12 December 2020

10.1016/S0022-510X(02)00421-5

Identifiers

DOI

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6 August 2017

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